Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis
Activity of renin-angiotensin-aldosterone and sympatho-adrenal systems (RAAS, SAS) in arterial hypertension combined with hypo- or hyperthyrosis was investigated. RAAS activity was decreased in hypothyrosis, and increased in hyperthyrosis; SAS activity was enhanced both in hypo- and hyperthyrosis. T...
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Format: | Article |
Language: | Russian |
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«FIRMA «SILICEA» LLC
2005-10-01
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Series: | Российский кардиологический журнал |
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Online Access: | https://russjcardiol.elpub.ru/jour/article/view/2359 |
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author | P. Kh. Dzhanashiya G. B. Selivanova |
author_facet | P. Kh. Dzhanashiya G. B. Selivanova |
author_sort | P. Kh. Dzhanashiya |
collection | DOAJ |
description | Activity of renin-angiotensin-aldosterone and sympatho-adrenal systems (RAAS, SAS) in arterial hypertension combined with hypo- or hyperthyrosis was investigated. RAAS activity was decreased in hypothyrosis, and increased in hyperthyrosis; SAS activity was enhanced both in hypo- and hyperthyrosis. These data correlate with the results of 24-hour blood pressure monitoring, and previous studies' results. The authors conclude that RAAS and SAS might play a role in AH pathogenesis among patients with hypo- or hyperthyrosis. |
first_indexed | 2024-04-09T20:54:47Z |
format | Article |
id | doaj.art-de10c046c7884a9b8cfed7b1b9e57126 |
institution | Directory Open Access Journal |
issn | 1560-4071 2618-7620 |
language | Russian |
last_indexed | 2024-04-09T20:54:47Z |
publishDate | 2005-10-01 |
publisher | «FIRMA «SILICEA» LLC |
record_format | Article |
series | Российский кардиологический журнал |
spelling | doaj.art-de10c046c7884a9b8cfed7b1b9e571262023-03-29T21:23:12Zrus«FIRMA «SILICEA» LLCРоссийский кардиологический журнал1560-40712618-76202005-10-010534392105Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosisP. Kh. Dzhanashiya0G. B. Selivanova1Российский государственный университетРоссийский государственный университетActivity of renin-angiotensin-aldosterone and sympatho-adrenal systems (RAAS, SAS) in arterial hypertension combined with hypo- or hyperthyrosis was investigated. RAAS activity was decreased in hypothyrosis, and increased in hyperthyrosis; SAS activity was enhanced both in hypo- and hyperthyrosis. These data correlate with the results of 24-hour blood pressure monitoring, and previous studies' results. The authors conclude that RAAS and SAS might play a role in AH pathogenesis among patients with hypo- or hyperthyrosis.https://russjcardiol.elpub.ru/jour/article/view/2359arterial hypertensionhypothyrosishyperthyrosisrenin-angiotensin-aldosterone systemsympatho-adrenal system |
spellingShingle | P. Kh. Dzhanashiya G. B. Selivanova Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis Российский кардиологический журнал arterial hypertension hypothyrosis hyperthyrosis renin-angiotensin-aldosterone system sympatho-adrenal system |
title | Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis |
title_full | Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis |
title_fullStr | Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis |
title_full_unstemmed | Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis |
title_short | Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis |
title_sort | neurohumoral activation in arterial hypertension pathogenesis among patients with hypod and hyperthyrosis |
topic | arterial hypertension hypothyrosis hyperthyrosis renin-angiotensin-aldosterone system sympatho-adrenal system |
url | https://russjcardiol.elpub.ru/jour/article/view/2359 |
work_keys_str_mv | AT pkhdzhanashiya neurohumoralactivationinarterialhypertensionpathogenesisamongpatientswithhypodandhyperthyrosis AT gbselivanova neurohumoralactivationinarterialhypertensionpathogenesisamongpatientswithhypodandhyperthyrosis |