Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis

Activity of renin-angiotensin-aldosterone and sympatho-adrenal systems (RAAS, SAS) in arterial hypertension combined with hypo- or hyperthyrosis was investigated. RAAS activity was decreased in hypothyrosis, and increased in hyperthyrosis; SAS activity was enhanced both in hypo- and hyperthyrosis. T...

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Main Authors: P. Kh. Dzhanashiya, G. B. Selivanova
Format: Article
Language:Russian
Published: «FIRMA «SILICEA» LLC 2005-10-01
Series:Российский кардиологический журнал
Subjects:
Online Access:https://russjcardiol.elpub.ru/jour/article/view/2359
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author P. Kh. Dzhanashiya
G. B. Selivanova
author_facet P. Kh. Dzhanashiya
G. B. Selivanova
author_sort P. Kh. Dzhanashiya
collection DOAJ
description Activity of renin-angiotensin-aldosterone and sympatho-adrenal systems (RAAS, SAS) in arterial hypertension combined with hypo- or hyperthyrosis was investigated. RAAS activity was decreased in hypothyrosis, and increased in hyperthyrosis; SAS activity was enhanced both in hypo- and hyperthyrosis. These data correlate with the results of 24-hour blood pressure monitoring, and previous studies' results. The authors conclude that RAAS and SAS might play a role in AH pathogenesis among patients with hypo- or hyperthyrosis.
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spelling doaj.art-de10c046c7884a9b8cfed7b1b9e571262023-03-29T21:23:12Zrus«FIRMA «SILICEA» LLCРоссийский кардиологический журнал1560-40712618-76202005-10-010534392105Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosisP. Kh. Dzhanashiya0G. B. Selivanova1Российский государственный университетРоссийский государственный университетActivity of renin-angiotensin-aldosterone and sympatho-adrenal systems (RAAS, SAS) in arterial hypertension combined with hypo- or hyperthyrosis was investigated. RAAS activity was decreased in hypothyrosis, and increased in hyperthyrosis; SAS activity was enhanced both in hypo- and hyperthyrosis. These data correlate with the results of 24-hour blood pressure monitoring, and previous studies' results. The authors conclude that RAAS and SAS might play a role in AH pathogenesis among patients with hypo- or hyperthyrosis.https://russjcardiol.elpub.ru/jour/article/view/2359arterial hypertensionhypothyrosishyperthyrosisrenin-angiotensin-aldosterone systemsympatho-adrenal system
spellingShingle P. Kh. Dzhanashiya
G. B. Selivanova
Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis
Российский кардиологический журнал
arterial hypertension
hypothyrosis
hyperthyrosis
renin-angiotensin-aldosterone system
sympatho-adrenal system
title Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis
title_full Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis
title_fullStr Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis
title_full_unstemmed Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis
title_short Neurohumoral activation in arterial hypertension pathogenesis among patients with hypoD and hyperthyrosis
title_sort neurohumoral activation in arterial hypertension pathogenesis among patients with hypod and hyperthyrosis
topic arterial hypertension
hypothyrosis
hyperthyrosis
renin-angiotensin-aldosterone system
sympatho-adrenal system
url https://russjcardiol.elpub.ru/jour/article/view/2359
work_keys_str_mv AT pkhdzhanashiya neurohumoralactivationinarterialhypertensionpathogenesisamongpatientswithhypodandhyperthyrosis
AT gbselivanova neurohumoralactivationinarterialhypertensionpathogenesisamongpatientswithhypodandhyperthyrosis