Absence of sodA Increases the Levels of Oxidation of Key Metabolic Determinants of Borrelia burgdorferi.
Borrelia burgdorferi, the causative agent of Lyme disease, alters its gene expression in response to environmental signals unique to its tick vector or vertebrate hosts. B. burgdorferi carries one superoxide dismutase gene (sodA) capable of controlling intracellular superoxide levels. Previously, so...
Main Authors: | Maria D Esteve-Gassent, Trever C Smith, Christina M Small, Derek P Thomas, J Seshu |
---|---|
Format: | Article |
Language: | English |
Published: |
Public Library of Science (PLoS)
2015-01-01
|
Series: | PLoS ONE |
Online Access: | http://europepmc.org/articles/PMC4556403?pdf=render |
Similar Items
-
Immunization with a Borrelia burgdorferi BB0172-derived peptide protects mice against lyme disease.
by: Christina M Small, et al.
Published: (2014-01-01) -
Effect of levels of acetate on the mevalonate pathway of Borrelia burgdorferi.
by: Tricia A Van Laar, et al.
Published: (2012-01-01) -
Borrelia peptidoglycan interacting Protein (BpiP) contributes to the fitness of Borrelia burgdorferi against host-derived factors and influences virulence in mouse models of Lyme disease.
by: Yue Chen, et al.
Published: (2021-04-01) -
Prevalencia de Borrelia burgdorferi sensu lato en roedores sinantrópicos de dos comunidades rurales de Yucatán, México
by: Analilia Solís-Hernández, et al.
Published: (2016-04-01) -
Pathogenicity and virulence of Borrelia burgdorferi
by: Martin Strnad, et al.
Published: (2023-12-01)