Arachidonic acid-induced Ca2+ entry and migration in a neuroendocrine cancer cell line
Abstract Background Store-operated Ca2+ entry (SOCE) has been implicated in the migration of some cancer cell lines. The canonical SOCE is defined as the Ca2+ entry that occurs in response to near-maximal depletion of Ca2+ within the endoplasmic reticulum. Alternatively, arachidonic acid (AA) has be...
Main Authors: | Priyodarshan Goswamee, Tamar Pounardjian, David R. Giovannucci |
---|---|
Format: | Article |
Language: | English |
Published: |
BMC
2018-03-01
|
Series: | Cancer Cell International |
Subjects: | |
Online Access: | http://link.springer.com/article/10.1186/s12935-018-0529-8 |
Similar Items
-
Arachidonic Acid Attenuates Cell Proliferation, Migration and Viability by a Mechanism Independent on Calcium Entry
by: Carlos Cantonero, et al.
Published: (2020-05-01) -
Transverse tubule remodeling enhances Orai1-dependent Ca2+ entry in skeletal muscle
by: Antonio Michelucci, et al.
Published: (2019-10-01) -
An essential and NSF independent role for α-SNAP in store-operated calcium entry
by: Yong Miao, et al.
Published: (2013-07-01) -
Orai1 Channels Are Essential for Amplification of Glutamate-Evoked Ca2+ Signals in Dendritic Spines to Regulate Working and Associative Memory
by: Mohammad Mehdi Maneshi, et al.
Published: (2020-12-01) -
TRPC1 channel clustering during store-operated Ca2+ entry in keratinocytes
by: Declan Manning, et al.
Published: (2023-03-01)