CDA gene silencing regulated the proliferation and apoptosis of chronic myeloid leukemia K562 cells
Abstract Background As a disease of hematopoietic stem cell, chronic myeloid leukemia (CML) possesses unique biological and clinical features. However, the biologic mechanism underlying its development remains poorly understood. Thus, the objective of the present study is to discuss the effect of cy...
المؤلفون الرئيسيون: | Xiao-Fang Wei, You-Fan Feng, Qiao-Lin Chen, Qi-Ke Zhang |
---|---|
التنسيق: | مقال |
اللغة: | English |
منشور في: |
BMC
2018-07-01
|
سلاسل: | Cancer Cell International |
الموضوعات: | |
الوصول للمادة أونلاين: | http://link.springer.com/article/10.1186/s12935-018-0587-y |
مواد مشابهة
-
Knockdown of NUDT21 inhibits proliferation and promotes apoptosis of human K562 leukemia cells through ERK pathway
حسب: Zhang L, وآخرون
منشور في: (2018-10-01) -
Interaction of xanthoxyletin with protein, molecular docking and antitumor activity in human chronic myeloid leukemia K562 cells
حسب: Chen Wang, وآخرون
منشور في: (2024-12-01) -
The role of CSE1L silencing in the regulation of proliferation and apoptosis via the AMPK/mTOR signaling pathway in chronic myeloid leukemia
حسب: Xiao-Yu Liu, وآخرون
منشور في: (2023-12-01) -
CDA deficiency is not associated with higher sensitivity to immune checkpoint inhibitors in melanoma patients
حسب: C. Gaudy-Marqueste, وآخرون
منشور في: (2024-12-01) -
Synergistic Effect of Dexamethasone and Diclofenac on Viability of K562 Cells
حسب: Hasan Tahazadeh, وآخرون
منشور في: (2022-07-01)