N-Acetylcysteine, an ROS Inhibitor, Alleviates the Pathophysiology of Hyperthyroidism-Induced Cardiomyopathy via the ROS/Ca<sup>2+</sup> Pathway

N-Acetylcysteine, an ROS Inhibitor, Alleviates the Pathophysiology of Hyperthyroidism-Induced Cardiomyopathy via the ROS/Ca<sup>2+</sup> Pathway

Hyperthyroidism is common and can induce cardiomyopathy, but there is no effective therapeutic strategy. The purpose of this study was to investigate the molecular mechanism of hyperthyroidism-induced cardiomyopathy (HTC) and the effect of N-acetylcysteine (NAC), an ROS inhibitor, on the pathophysio...

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Bibliographic Details
Main Authors: Mengni Bao, Xiumeng Hua, Han Mo, Zhe Sun, Bo Xu, Xiao Chen, Mengda Xu, Xinjie Xu, Jiangping Song
Format: Article
Language:English
Published: MDPI AG 2022-08-01
Series:Biomolecules
Subjects:
hyperthyroidism-induced cardiomyopathy
NAC
ROS/Ca<sup>2+</sup>
fibrosis
apoptosis
hypertrophy
Online Access:https://www.mdpi.com/2218-273X/12/9/1195

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