SalA Attenuates Hypoxia-Induced Endothelial Endoplasmic Reticulum Stress and Apoptosis via Down-Regulation of VLDL Receptor Expression

SalA Attenuates Hypoxia-Induced Endothelial Endoplasmic Reticulum Stress and Apoptosis via Down-Regulation of VLDL Receptor Expression

Background: Salvianolic acid A (SalA) has been shown to display robust protection against endothelial injury. VLDL receptor (VLDLr) is expressed at high levels in the endothelial cells. However its endothelial biological function has not been completely elucidated. Here, we investigated molecular ef...

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Bibliographic Details
Main Authors: Ping Xie, Yingchun Duan, Xianzhi Guo, Lina Hu, Minghua Yu
Format: Article
Language:English
Published: Cell Physiol Biochem Press GmbH & Co KG 2015-01-01
Series:Cellular Physiology and Biochemistry
Subjects:
Apoptosis
Salvianolic acid A
Hypoxia
VLDL receptor
Endothelial endoplasmic reticulum stress
Online Access:http://www.karger.com/Article/FullText/369671

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