SARS-CoV-2 S1 protein causes brain inflammation by reducing intracerebral acetylcholine production
Summary: Neurological complications that occur in SARS-CoV-2 infection, such as olfactory dysfunction, brain inflammation, malaise, and depressive symptoms, are thought to contribute to long COVID. However, in autopsies of patients who have died from COVID-19, there is normally no direct evidence th...
Main Authors: | Naomi Oka, Kazuya Shimada, Azusa Ishii, Nobuyuki Kobayashi, Kazuhiro Kondo |
---|---|
Format: | Article |
Language: | English |
Published: |
Elsevier
2023-06-01
|
Series: | iScience |
Subjects: | |
Online Access: | http://www.sciencedirect.com/science/article/pii/S2589004223010313 |
Similar Items
-
Identification of a strong genetic risk factor for major depressive disorder in the human virome
by: Nobuyuki Kobayashi, et al.
Published: (2024-03-01) -
Human Herpesvirus 6B Greatly Increases Risk of Depression by Activating Hypothalamic-Pituitary -Adrenal Axis during Latent Phase of Infection
by: Nobuyuki Kobayashi, et al.
Published: (2020-06-01) -
Human iPS cell-derived sensory neurons can be infected by SARS-CoV-2
by: Anthony Flamier, et al.
Published: (2023-09-01) -
Immunosuppression-induced Zika virus reactivation causes brain inflammation and behavioral deficits in mice
by: Clara de O. Nogueira, et al.
Published: (2024-07-01) -
Temperature and sex shape Zika virus pathogenicity in the adult Bratcheesehead brain: A Drosophila model for virus-associated neurological diseases
by: Ghada Tafesh-Edwards, et al.
Published: (2023-04-01)