Urovni rezistina, adiponektinai insulinorezistentnosti u patsientov s raznoystepen'yu narusheniy uglevodnogo obmena
The basis of type 2 diabetes is obesity and insulin resistance. Fat tissue is hormonaly active system which produces adiponectin, resistin and other adipokines. These substances participate in development of insulin resistance. The article presents the results of a study conducted to examine the rol...
| Format: | Article |
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| Language: | English |
| Published: |
Endocrinology Research Centre
2011-09-01
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| Series: | Ожирение и метаболизм |
| Subjects: | |
| Online Access: | https://www.omet-endojournals.ru/jour/article/view/4838 |
| _version_ | 1827571913502752768 |
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| collection | DOAJ |
| description | The basis of type 2 diabetes is obesity and insulin resistance. Fat tissue is hormonaly active system which produces adiponectin, resistin and other adipokines. These substances participate in development of insulin resistance. The article presents the results of a study conducted to examine the role of adiponectin, resistin in development of insulin resistance in patients with IGT and type 2 diabetes. The reduction of adiponectin level and insulin resistance was found in women suffering from IGF or type 2 diabetes and in men with only type 2 diabetes. Resistin levels were increased in the examined patients with IGT and with type 2 diabetes. |
| first_indexed | 2024-03-08T09:14:44Z |
| format | Article |
| id | doaj.art-f62d2817f7fc447bbf2f57de83a98964 |
| institution | Directory Open Access Journal |
| issn | 2071-8713 2306-5524 |
| language | English |
| last_indexed | 2025-03-20T13:59:02Z |
| publishDate | 2011-09-01 |
| publisher | Endocrinology Research Centre |
| record_format | Article |
| series | Ожирение и метаболизм |
| spelling | doaj.art-f62d2817f7fc447bbf2f57de83a989642024-09-11T16:17:47ZengEndocrinology Research CentreОжирение и метаболизм2071-87132306-55242011-09-0183576010.14341/2071-8713-48384796Urovni rezistina, adiponektinai insulinorezistentnosti u patsientov s raznoystepen'yu narusheniy uglevodnogo obmenaThe basis of type 2 diabetes is obesity and insulin resistance. Fat tissue is hormonaly active system which produces adiponectin, resistin and other adipokines. These substances participate in development of insulin resistance. The article presents the results of a study conducted to examine the role of adiponectin, resistin in development of insulin resistance in patients with IGT and type 2 diabetes. The reduction of adiponectin level and insulin resistance was found in women suffering from IGF or type 2 diabetes and in men with only type 2 diabetes. Resistin levels were increased in the examined patients with IGT and with type 2 diabetes.https://www.omet-endojournals.ru/jour/article/view/4838diabetes mellitus type 2impaired glucose toleranceinsulin resistanceinsulinadiponectinresistin |
| spellingShingle | Urovni rezistina, adiponektinai insulinorezistentnosti u patsientov s raznoystepen'yu narusheniy uglevodnogo obmena Ожирение и метаболизм diabetes mellitus type 2 impaired glucose tolerance insulin resistance insulin adiponectin resistin |
| title | Urovni rezistina, adiponektinai insulinorezistentnosti u patsientov s raznoystepen'yu narusheniy uglevodnogo obmena |
| title_full | Urovni rezistina, adiponektinai insulinorezistentnosti u patsientov s raznoystepen'yu narusheniy uglevodnogo obmena |
| title_fullStr | Urovni rezistina, adiponektinai insulinorezistentnosti u patsientov s raznoystepen'yu narusheniy uglevodnogo obmena |
| title_full_unstemmed | Urovni rezistina, adiponektinai insulinorezistentnosti u patsientov s raznoystepen'yu narusheniy uglevodnogo obmena |
| title_short | Urovni rezistina, adiponektinai insulinorezistentnosti u patsientov s raznoystepen'yu narusheniy uglevodnogo obmena |
| title_sort | urovni rezistina adiponektinai insulinorezistentnosti u patsientov s raznoystepen yu narusheniy uglevodnogo obmena |
| topic | diabetes mellitus type 2 impaired glucose tolerance insulin resistance insulin adiponectin resistin |
| url | https://www.omet-endojournals.ru/jour/article/view/4838 |