Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess

Abstract A 61‐year‐old male presented with progressive generalized weakness, myalgia, diaphoresis, fever, episodic chills and rigors that had started 4 days previously. Chest x‐ray (CXR) showed overlying curvilinear radio‐opacities. Abdominal computed tomography revealed liver and bilateral adrenal...

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Main Authors: Haroon Chaudhry, Swathi Nimmala, Bhavani Nagendra Papudesi, Fizza Sajjad, Sanu Paul, Zimran Gohar, Reuben Azad, Hannah Naveen, Joseph Demidovich
Format: Article
Language:English
Published: Wiley 2021-09-01
Series:Respirology Case Reports
Subjects:
Online Access:https://doi.org/10.1002/rcr2.826
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author Haroon Chaudhry
Swathi Nimmala
Bhavani Nagendra Papudesi
Fizza Sajjad
Sanu Paul
Zimran Gohar
Reuben Azad
Hannah Naveen
Joseph Demidovich
author_facet Haroon Chaudhry
Swathi Nimmala
Bhavani Nagendra Papudesi
Fizza Sajjad
Sanu Paul
Zimran Gohar
Reuben Azad
Hannah Naveen
Joseph Demidovich
author_sort Haroon Chaudhry
collection DOAJ
description Abstract A 61‐year‐old male presented with progressive generalized weakness, myalgia, diaphoresis, fever, episodic chills and rigors that had started 4 days previously. Chest x‐ray (CXR) showed overlying curvilinear radio‐opacities. Abdominal computed tomography revealed liver and bilateral adrenal lesions. Empiric 7‐day intravenous Piperacillin / Tazobactam (Zosyn) was initiated, and he was admitted for sepsis. After an episode of rigors on Day 2, he developed acute hypoxic respiratory failure with inspiratory stridor. CXR revealed new, bilateral airspace disease. Racemic Epinephrine, Solumedrol, Ketorolac (Toradol) and Diphenhydramine were given, and he was transferred to the intensive care unit with presumptive diagnosis of foreign body aspiration or allergic reaction. With each subsequent episode of rigor and chills, he continued developing hypoxic respiratory failure with stridor and an incremental increase in pulmonary oedema on imaging. Pulmonologist concluded it likely secondary to negative pressure pulmonary oedema caused by transient laryngeal dyskinesia induced by the increased work of breathing associated with rigors. Symptoms resolved after the complete course of antibiotics along with supportive therapy.
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spelling doaj.art-f78d5f20511d4b14b260635ddc128a612022-12-21T20:15:48ZengWileyRespirology Case Reports2051-33802021-09-0199n/an/a10.1002/rcr2.826Negative pressure pulmonary oedema due to rigors and chills associated with liver abscessHaroon Chaudhry0Swathi Nimmala1Bhavani Nagendra Papudesi2Fizza Sajjad3Sanu Paul4Zimran Gohar5Reuben Azad6Hannah Naveen7Joseph Demidovich8Department of Internal Medicine Suburban Community Hospital East Norriton Pennsylvania USADepartment of Internal Medicine Suburban Community Hospital East Norriton Pennsylvania USADepartment of Internal Medicine Suburban Community Hospital East Norriton Pennsylvania USADepartment of Science University of Albany Albany New York USADepartment of Internal Medicine Suburban Community Hospital East Norriton Pennsylvania USADepartment of Internal Medicine Suburban Community Hospital East Norriton Pennsylvania USADepartment of Cardiology Albert Einstein Medical Center Philadelphia Pennsylvania USADepartment of Medicine All Saints University School of Medicine Roseau DominicaDepartment of Internal Medicine Suburban Community Hospital East Norriton Pennsylvania USAAbstract A 61‐year‐old male presented with progressive generalized weakness, myalgia, diaphoresis, fever, episodic chills and rigors that had started 4 days previously. Chest x‐ray (CXR) showed overlying curvilinear radio‐opacities. Abdominal computed tomography revealed liver and bilateral adrenal lesions. Empiric 7‐day intravenous Piperacillin / Tazobactam (Zosyn) was initiated, and he was admitted for sepsis. After an episode of rigors on Day 2, he developed acute hypoxic respiratory failure with inspiratory stridor. CXR revealed new, bilateral airspace disease. Racemic Epinephrine, Solumedrol, Ketorolac (Toradol) and Diphenhydramine were given, and he was transferred to the intensive care unit with presumptive diagnosis of foreign body aspiration or allergic reaction. With each subsequent episode of rigor and chills, he continued developing hypoxic respiratory failure with stridor and an incremental increase in pulmonary oedema on imaging. Pulmonologist concluded it likely secondary to negative pressure pulmonary oedema caused by transient laryngeal dyskinesia induced by the increased work of breathing associated with rigors. Symptoms resolved after the complete course of antibiotics along with supportive therapy.https://doi.org/10.1002/rcr2.826critical caredyspnoealaryngeal dyskinesialiver abscessNPPE
spellingShingle Haroon Chaudhry
Swathi Nimmala
Bhavani Nagendra Papudesi
Fizza Sajjad
Sanu Paul
Zimran Gohar
Reuben Azad
Hannah Naveen
Joseph Demidovich
Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess
Respirology Case Reports
critical care
dyspnoea
laryngeal dyskinesia
liver abscess
NPPE
title Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess
title_full Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess
title_fullStr Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess
title_full_unstemmed Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess
title_short Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess
title_sort negative pressure pulmonary oedema due to rigors and chills associated with liver abscess
topic critical care
dyspnoea
laryngeal dyskinesia
liver abscess
NPPE
url https://doi.org/10.1002/rcr2.826
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