Favorable outcomes of NPM1mut AML patients are due to transcriptional inactivation of FOXM1, presenting a new target to overcome chemoresistance

Bibliographic Details
Main Authors: I. Khan, A. Kaempf, S. Raghuwanshi, M. Chesnokov, X. Zhang, Z. Wang, A. Domling, J. W. Tyner, C. Camacho, A. L. Gartel
Format: Article
Language:English
Published: Nature Publishing Group 2023-08-01
Series:Blood Cancer Journal
Online Access:https://doi.org/10.1038/s41408-023-00898-4
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author I. Khan
A. Kaempf
S. Raghuwanshi
M. Chesnokov
X. Zhang
Z. Wang
A. Domling
J. W. Tyner
C. Camacho
A. L. Gartel
author_facet I. Khan
A. Kaempf
S. Raghuwanshi
M. Chesnokov
X. Zhang
Z. Wang
A. Domling
J. W. Tyner
C. Camacho
A. L. Gartel
author_sort I. Khan
collection DOAJ
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issn 2044-5385
language English
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publishDate 2023-08-01
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record_format Article
series Blood Cancer Journal
spelling doaj.art-f9819aaa5aff4a02970deed6557e6a422023-11-19T12:25:32ZengNature Publishing GroupBlood Cancer Journal2044-53852023-08-011311510.1038/s41408-023-00898-4Favorable outcomes of NPM1mut AML patients are due to transcriptional inactivation of FOXM1, presenting a new target to overcome chemoresistanceI. Khan0A. Kaempf1S. Raghuwanshi2M. Chesnokov3X. Zhang4Z. Wang5A. Domling6J. W. Tyner7C. Camacho8A. L. Gartel9University of Illinois at Chicago, Department of MedicineOHSU Knight Cancer Institute, School of MedicineUniversity of Illinois at Chicago, Department of MedicineCentro Nacional de Investigaciones Oncológicas (CNIO)University of Illinois at Chicago, Department of MedicineThe Czech Advanced Technology and Research Institute (CATRIN) of Palacký UniversityThe Czech Advanced Technology and Research Institute (CATRIN) of Palacký UniversityOHSU Knight Cancer Institute, School of MedicineComputational and Systems Biology, University of PittsburghUniversity of Illinois at Chicago, Department of Medicinehttps://doi.org/10.1038/s41408-023-00898-4
spellingShingle I. Khan
A. Kaempf
S. Raghuwanshi
M. Chesnokov
X. Zhang
Z. Wang
A. Domling
J. W. Tyner
C. Camacho
A. L. Gartel
Favorable outcomes of NPM1mut AML patients are due to transcriptional inactivation of FOXM1, presenting a new target to overcome chemoresistance
Blood Cancer Journal
title Favorable outcomes of NPM1mut AML patients are due to transcriptional inactivation of FOXM1, presenting a new target to overcome chemoresistance
title_full Favorable outcomes of NPM1mut AML patients are due to transcriptional inactivation of FOXM1, presenting a new target to overcome chemoresistance
title_fullStr Favorable outcomes of NPM1mut AML patients are due to transcriptional inactivation of FOXM1, presenting a new target to overcome chemoresistance
title_full_unstemmed Favorable outcomes of NPM1mut AML patients are due to transcriptional inactivation of FOXM1, presenting a new target to overcome chemoresistance
title_short Favorable outcomes of NPM1mut AML patients are due to transcriptional inactivation of FOXM1, presenting a new target to overcome chemoresistance
title_sort favorable outcomes of npm1mut aml patients are due to transcriptional inactivation of foxm1 presenting a new target to overcome chemoresistance
url https://doi.org/10.1038/s41408-023-00898-4
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