Genetic instability is not a requirement for tumor development.

Genetic instability is not a requirement for tumor development.

Εμφάνιση άλλων εκδόσεων (1)

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριος συγγραφέας:	Bodmer, W
Μορφή:	Journal article
Γλώσσα:	English
Έκδοση:	2008

Παρόμοια τεκμήρια

Genetic instability is not a requirement for tumor development.
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Έκδοση: (2008)

Genetic instability is not a requirement for tumor development (vol 68, pg 3558, 2008)
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Expression of Interleukin-8 Promotes Neutrophil Infiltration and Genetic Instability in Mutatect Tumors
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Genetic evidence that SMAD2 is not required for gonadal tumor development in inhibin-deficient mice
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Familial Adenomatous Polyposis: Analysis of Genetic Instability of Microsatellites Loci and Genetic Alternations of Tumor Suppressor Genes
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SOMATIC-CELL GENETICS
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The Role of Tumor Microenvironment in Genomic Instability of Malignant Tumors
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Plk1 overexpression induces chromosomal instability and suppresses tumor development
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PTEN C-Terminal Deletion Causes Genomic Instability and Tumor Development
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A race between tumor immunoescape and genome maintenance selects for optimum levels of (epi)genetic instability.
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Tumor Hypoxia Drives Genomic Instability
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SMAD4 mutations in colorectal cancer probably occur before chromosomal instability, but after divergence of the microsatellite instability pathway.
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SMAD4 mutations in colorectal cancer probably occur before chromosomal instability, but after divergence of the microsatellite instability pathway
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Population genetics of tumours.
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Allele loss occurs frequently at hMLH1, but rarely at hMSH2, in sporadic colorectal cancers with microsatellite instability.
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Genetic Aspects of Gastric Cancer Instability
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Genetic instability in cyanobacteria – an elephant in the room?
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Genetic and Epigenetic Instability of Stem Cells
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DNA repair, genetic instability, and cancer /
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Genomic instability as a driver and suppressor of anti-tumor immunity
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Genetic instability as a driver for immune surveillance
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Genetic factors in Hodgkin's disease: association with a disease-susceptibility locus (DSA) in the HL-A region.
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Werner Syndrome Helicase Is Required for the Survival of Cancer Cells with Microsatellite Instability
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