Evaluation of the role of miR-31-dependent reduction in dystrophin and nNOS on atrial-fibrillation-induced electrical remodelling in man
The management of atrial fibrillation remains a challenge. This condition remodels atrial electrical properties, which promote resistance to treatment. Although remodelling has long been a therapeutic target in atrial fibrillation, its causes remain incompletely understood. We aimed to evaluate the...
Príomhchruthaitheoirí: | Reilly, S, Liu, X, Carnicer, R, Rajakumar, T, Sayeed, R, Krasopoulos, G, Verheule, S, Fulga, T, Schotten, U, Casadei, B |
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Formáid: | Conference item |
Teanga: | English |
Foilsithe / Cruthaithe: |
Elsevier
2015
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Míreanna comhchosúla
Míreanna comhchosúla
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Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase
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Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase.
de réir: Reilly, S, et al.
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Atrial Fibrillation Causes a Progressive, Time-Dependent Reduction in Myocardial Neuronal NOS in Humans and Goats: Implications for AF-Induced Electrical Remodeling
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Time-dependent regional differences in sources of oxidative stress in atrial fibrillation-induced remodelling
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ATRIAL SOURCES OF REACTIVE OXYGEN SPECIES VARY WITH THE SUBSTRATE AND DURATION OF ATRIAL FIBRILLATION: IMPLICATIONS FOR THE ANTIARRHYTHMIC EFFECT OF STATINS
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