Evaluation of the role of miR-31-dependent reduction in dystrophin and nNOS on atrial-fibrillation-induced electrical remodelling in man
The management of atrial fibrillation remains a challenge. This condition remodels atrial electrical properties, which promote resistance to treatment. Although remodelling has long been a therapeutic target in atrial fibrillation, its causes remain incompletely understood. We aimed to evaluate the...
मुख्य लेखकों: | Reilly, S, Liu, X, Carnicer, R, Rajakumar, T, Sayeed, R, Krasopoulos, G, Verheule, S, Fulga, T, Schotten, U, Casadei, B |
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स्वरूप: | Conference item |
भाषा: | English |
प्रकाशित: |
Elsevier
2015
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समान संसाधन
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Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase
द्वारा: Reilly, S, और अन्य
प्रकाशित: (2016) -
Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase.
द्वारा: Reilly, S, और अन्य
प्रकाशित: (2016) -
Atrial Fibrillation Causes a Progressive, Time-Dependent Reduction in Myocardial Neuronal NOS in Humans and Goats: Implications for AF-Induced Electrical Remodeling
द्वारा: Reilly, S, और अन्य
प्रकाशित: (2012) -
Time-dependent regional differences in sources of oxidative stress in atrial fibrillation-induced remodelling
द्वारा: Reilly, SN, और अन्य
प्रकाशित: (2008) -
ATRIAL SOURCES OF REACTIVE OXYGEN SPECIES VARY WITH THE SUBSTRATE AND DURATION OF ATRIAL FIBRILLATION: IMPLICATIONS FOR THE ANTIARRHYTHMIC EFFECT OF STATINS
द्वारा: Reilly, S, और अन्य
प्रकाशित: (2011)