The importance of the bone marrow microenvironment in multiple myeloma; Differential responses to lithium in vivo

The importance of the bone marrow microenvironment in multiple myeloma; Differential responses to lithium in vivo

Chi tiết về thư mục
Những tác giả chính:	Edwards, C, Mundy, G
Định dạng:	Conference item
Được phát hành:	2006

Những quyển sách tương tự

The Bone Marrow Microenvironment in Multiple Myeloma
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Myeloma cells exhibit an increase in proteasome activity and an enhanced response to proteasome inhibition in the bone marrow microenvironment in vivo.
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Increasing Wnt signaling in the bone marrow microenvironment inhibits the development of myeloma bone disease and reduces tumor burden in bone in vivo.
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Lithium inhibits the development of myeloma bone disease in vivo.
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Targeting Wnt signaling in myeloma in vivo; Differential effects on tumor burden and myeloma bone disease
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The ubiquitin-proteasome pathway is dysregulated in myeloma cells in the bone microenvironment in vivo
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Bone marrow stromal cells create a permissive microenvironment for myeloma development: a new stromal role for Wnt inhibitor Dkk1.
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The pathogenesis of the bone disease of multiple myeloma.
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In vivo bone marrow microenvironment siRNA delivery using lipid–polymer nanoparticles for multiple myeloma therapy
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Host Bone Marrow-Derived Stromal Cells Promote Myeloma Initiation and Development of Osteolysis
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Osteoclasts in Myeloma Are Derived from Gr-1(+)CD11b(+) Myeloid Immune Suppressor Cells of the Bone Marrow Niche in Vivo
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Excessive serine from the bone marrow microenvironment impairs megakaryopoiesis and thrombopoiesis in Multiple Myeloma
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Adipokines, adiposity, and bone marrow adipocytes: Dangerous accomplices in multiple myeloma
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Osteoclasts in myeloma are derived from Gr-1+CD11b+mononuclear cells of the bone marrow niche
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Myeloma cells decrease EphB4 expression in osteoblasts; A novel mechanism for regulation of bone formation in multiple myeloma
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Myeloma cells decrease EphB4 expression in osteoblasts: A novel mechanism for regulation of bone formation in multiple myeloma
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The role of bone marrow microenvironment in the progression of multiple myeloma from monoclonal gammopathy of undetermined significance
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Autophagy and the Bone Marrow Microenvironment: A Review of Protective Factors in the Development and Maintenance of Multiple Myeloma
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Agonists of TRAIL death receptors induce myeloma cell apoptosis that is not prevented by cells of the bone marrow microenvironment.
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The KISS1 Receptor as an In Vivo Microenvironment Imaging Biomarker of Multiple Myeloma Bone Disease.
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Increasing adiponectin via an apolipoprotein peptide mimetic, L-4F, increases bone formation in normal mice and prevents myeloma bone disease in vivo
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Extracellular vesicles mediate the communication between multiple myeloma and bone marrow microenvironment in a NOTCH dependent way
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Multiple myeloma hinders erythropoiesis and causes anaemia owing to high levels of CCL3 in the bone marrow microenvironment
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The antidiabetic drug metformin acts on the bone microenvironment to promote myeloma cell adhesion to preosteoblasts and increase myeloma tumour burden in vivo
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PD-L1/PD-1 Pattern of Expression Within the Bone Marrow Immune Microenvironment in Smoldering Myeloma and Active Multiple Myeloma Patients
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The antidiabetic drug metformin acts on the bone microenvironment to promote myeloma cell adhesion to preosteoblasts and increase myeloma tumour burden in vivo
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