GLUCOSE DOES NOT BLOCK ATP-REGULATED K+-CHANNEL ACTIVITY IN FETAL B-CELLS

GLUCOSE DOES NOT BLOCK ATP-REGULATED K+-CHANNEL ACTIVITY IN FETAL B-CELLS

Dettagli Bibliografici
Autori principali:	Welsh, M, Berggren, P, Rorsman, P
Natura:	Journal article
Pubblicazione:	1987

Documenti analoghi

DEFECTIVE REGULATION OF GLUCOSE-REGULATED AND ATP-REGULATED K-CHANNELS RESULTS IN DISTURBED GLUCOSE SENSITIVITY IN FETAL B-CELLS
di: Rorsman, P, et al.
Pubblicazione: (1988)

Failure of glucose to elicit a normal secretory response in fetal pancreatic beta cells results from glucose insensitivity of the ATP-regulated K+ channels.
di: Rorsman, P, et al.
Pubblicazione: (1989)

ATP-REGULATED K+ CHANNELS AND DIABETES-MELLITUS
di: Rorsman, P, et al.
Pubblicazione: (1990)

Does galanin inhibit insulin secretion by opening of the ATP-regulated K+ channel in the beta-cell?
di: Ahrén, B, et al.
Pubblicazione: (1989)

K(ATP)-channels and glucose-regulated glucagon secretion.
di: Rorsman, P, et al.
Pubblicazione: (2008)

Glucose dependent K+-channels in pancreatic beta-cells are regulated by intracellular ATP.
di: Rorsman, P, et al.
Pubblicazione: (1985)

Does the K-ATP channel participate in the regulation of glucagon secretion?
di: Bokvist, K, et al.
Pubblicazione: (1999)

ATP-sensitive K+ channel-dependent regulation of glucagon release and electrical activity by glucose in wild-type and SUR1-/- mouse alpha-cells.
di: Gromada, J, et al.
Pubblicazione: (2004)

Block of ATP-regulated and Ca2(+)-activated K+ channels in mouse pancreatic beta-cells by external tetraethylammonium and quinine.
di: Bokvist, K, et al.
Pubblicazione: (1990)

Glucose-dependent regulation of rhythmic action potential firing in pancreatic beta-cells by K(ATP)-channel modulation.
di: Kanno, T, et al.
Pubblicazione: (2002)

Inhibition of ATP-regulated K+ channels precedes depolarization-induced increase in cytoplasmic free Ca2+ concentration in pancreatic beta-cells.
di: Arkhammar, P, et al.
Pubblicazione: (1987)

EVIDENCE THAT GLUCOSE-INDUCED DEPOLARIZATION IN THE PANCREATIC BETA-CELLS INVOLVES THE CLOSURE OF AN ATP-DEPENDENT K+-CHANNEL
di: Rorsman, P, et al.
Pubblicazione: (1985)

Inhibition of ATP-regulated K(+)-channels by a photoactivatable ATP-analogue in mouse pancreatic beta-cells.
di: Ammälä, C, et al.
Pubblicazione: (1991)

Separate processes mediate nucleotide-induced inhibition and stimulation of the ATP-regulated K(+)-channels in mouse pancreatic beta-cells.
di: Bokvist, K, et al.
Pubblicazione: (1991)

EVIDENCE THAT ADP AND ATP BIND AT DIFFERENT SITES AT THE G-CHANNEL
di: Bokvist, K, et al.
Pubblicazione: (1988)

ATP-REGULATED K CHANNELS MODULATE ELECTRICAL-ACTIVITY IN MOUSE ISOLATED PANCREATIC BETA-CELLS
di: Ashcroft, F, et al.
Pubblicazione: (1990)

Simultaneous recordings of glucose dependent electrical activity and ATP-regulated K(+)-currents in isolated mouse pancreatic beta-cells.
di: Smith, P, et al.
Pubblicazione: (1990)

ATP-sensitive K+ channels: a link between B-cell metabolism and insulin secretion.
di: Ashcroft, F, et al.
Pubblicazione: (1990)

K(ATP) channels and islet hormone secretion: new insights and controversies.
di: Ashcroft, F, et al.
Pubblicazione: (2013)

GLUCOSE REGULATION OF GLUCAGON-SECRETION INVOLVES ACTIVATION OF GABAA-RECEPTOR CL--CHANNELS
di: Rorsman, P, et al.
Pubblicazione: (1989)

Phentolamine block of ATP-sensitive K+ channels does not involve interaction with the sulphonylurea receptor, SUR1.
di: Proks, P, et al.
Pubblicazione: (1997)

Characterisation of sulphonylurea and ATP-regulated K+ channels in rat pancreatic A-cells.
di: Bokvist, K, et al.
Pubblicazione: (1999)

Evidence from in situ human alpha-cells supporting intrinsic K-ATP channel-dependent regulation of glucagon secretion
di: Macdonald, P, et al.
Pubblicazione: (2006)

Demonstration of a novel apamin-insensitive calcium-activated K+ channel in mouse pancreatic B cells.
di: Ammälä, C, et al.
Pubblicazione: (1993)

Modeling K(ATP) channel gating and its regulation.
di: Proks, P, et al.
Pubblicazione: (2009)

Mechanism of K-ATP channel inhibition by ATP
di: Ashcroft, F, et al.
Pubblicazione: (1998)

Analysis of the differential modulation of sulphonylurea block of beta-cell and cardiac ATP-sensitive K+ (K(ATP)) channels by Mg-nucleotides.
di: Reimann, F, et al.
Pubblicazione: (2003)

Regulation of K-ATP channels by nucleotides and drugs
di: Gribble, F, et al.
Pubblicazione: (2000)

Differential metabolic and nucleotide sensitivity of beta-cell and cardiac K-ATP channels
di: Vedovato, N, et al.
Pubblicazione: (2018)

Opposite effects of tolbutamide and diazoxide on the ATP-dependent K+ channel in mouse pancreatic beta-cells.
di: Trube, G, et al.
Pubblicazione: (1986)

How ATP inhibits the open K(ATP) channel.
di: Craig, T, et al.
Pubblicazione: (2008)

Sulfonylurea-mediated stimulation of insulin exocytosis via an ATP-sensitive K+ channel-independent action.
di: Renström, E, et al.
Pubblicazione: (2002)

ATP-regulated potassium channels and voltage-gated calcium channels in pancreatic alpha and beta cells: Similar functions but reciprocal effects on secretion
di: Rorsman, P, et al.
Pubblicazione: (2014)

ATP-regulated potassium channels and voltage-gated calcium channels in pancreatic alpha and beta cells: similar functions but reciprocal effects on secretion.
di: Rorsman, P, et al.
Pubblicazione: (2014)

Molecular mechanism of sulphonylurea block of K(ATP) channels carrying mutations that impair ATP inhibition and cause neonatal diabetes.
di: Proks, P, et al.
Pubblicazione: (2013)

Activation by adrenaline of a low-conductance G protein-dependent K+ channel in mouse pancreatic B cells.
di: Rorsman, P, et al.
Pubblicazione: (1991)

Glucose-inhibition of glucagon secretion involves activation of GABAA-receptor chloride channels.
di: Rorsman, P, et al.
Pubblicazione: (1989)

Dynamic sensitivity of K-ATP channels to ATP
di: Loussouarn, G, et al.
Pubblicazione: (2001)

ADP MODULATES THE ATP-SENSITIVITY OF THE K-CHANNEL CLOSED BY GLUCOSE IN ISOLATED RAT PANCREATIC B-CELLS
di: Ashcroft, F, et al.
Pubblicazione: (1986)

Visualizing adenine nucleotide regulation of the K-ATP channel
di: Usher, SG, et al.
Pubblicazione: (2018)