HCM and DCM mutations in thin filament proteins have opposite effects on regulation of contractility in vitro

Podobne zapisy

• HCM and DCM mutations in cardiac thin filament proteins have opposite effects on the regulation of contractility in vitro
  od: Redwood, C, i wsp.
  Wydane: (2003)
• HCM and DCM mutations in thin filament proteins have opposite effects on in vitro motility
  od: Marston, S, i wsp.
  Wydane: (2002)
• Effects of DCM mutations in thin filament regulatory proteins on contractile function
  od: Robinson, P, i wsp.
  Wydane: (2005)
• In vitro motility studies of HCM and DCM mutations in cardiac muscle actin
  od: Dyer, E, i wsp.
  Wydane: (2007)
• Mutations in tropomyosin that cause DCM, affect cooperativity of cardiac muscle thin filaments
  od: Marston, S, i wsp.
  Wydane: (2007)