Hepatic CC chemokines control the magnitude of the inflammatory response within the injured rodent brain
Main Authors: | Campbell, S, Perry, V, Pitossi, F, Butchart, A, Chertoff, M, Waters, S, Anthony, D |
---|---|
Format: | Conference item |
Published: |
2004
|
Similar Items
-
Central nervous system injury triggers hepatic CC and CXC chemokine expression that is associated with leukocyte mobilization and recruitment to both the central nervous system and the liver.
by: Campbell, S, et al.
Published: (2005) -
Liver Kupffer cells control the magnitude of the inflammatory response in the injured brain and spinal cord.
by: Campbell, S, et al.
Published: (2008) -
Hypoxia Alters the Expression of CC Chemokines and CC Chemokine Receptors in a Tumor–A Literature Review
by: Jan Korbecki, et al.
Published: (2020-08-01) -
Overexpression of IL-1beta by adenoviral-mediated gene transfer in the rat brain causes a prolonged hepatic chemokine response, axonal injury and the suppression of spontaneous behaviour.
by: Campbell, S, et al.
Published: (2007) -
Altered chemokine expression in the spinal cord and brain contributes to differential interleukin-1beta-induced neutrophil recruitment.
by: Campbell, S, et al.
Published: (2002)