PROTECTIVE EFFECTS OF TETRAHYDROBIOPTERIN IN ISCHEMIA REPERFUSION INJURY DO NOT DEPEND ON DIMERISATION STATUS OF ENOS

PROTECTIVE EFFECTS OF TETRAHYDROBIOPTERIN IN ISCHEMIA REPERFUSION INJURY DO NOT DEPEND ON DIMERISATION STATUS OF ENOS

Bibliográfalaš dieđut
Váldodahkkit:	Maglione, M, Hein, S, Crabtree, M, Watschinger, K, Cardini, B, Oberhuber, R, Pratschke, J, Werner, E
Materiálatiipa:	Journal article
Almmustuhtton:	2013

Geahča maid

Endothelial nitric oxide synthase dysfunction in diabetic mice: importance of tetrahydrobiopterin in eNOS dimerisation.
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The Role of Tetrahydrobiopterin and Dihydrobiopterin in Ischemia/Reperfusion Injury When Given at Reperfusion
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Endothelial cell-specific roles for tetrahydrobiopterin in myocardial function, cardiac hypertrophy, and response to myocardial ischemia-reperfusion injury
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Tetrahydrobiopterin- and glutathionylation-dependent regulation of eNOS coupling: Relationships revealed by eNOS glutathionylation mutants in cells with tet-regulated GTP cyclohydrolase I expression
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Ischemia/Reperfusion Injury, Its Mechanisms, and Prevention
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A pivotal role for tryptophan 447 in enzymatic coupling of human endothelial nitric oxide synthase (eNOS): effects on tetrahydrobiopterin-dependent catalysis and eNOS dimerization.
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Tetrahydrobiopterin availability directly regulates eNOS coupling in tet-regulatable cells
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Regulation of eNOS function by tetrahydrobiopterin in vascular disease
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Myocardial protection by co-administration of l-arginine and tetrahydrobiopterin during ischemia and reperfusion
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Myocardial protection by co-administration of L-arginine and tetrahydrobiopterin during ischemia and reperfusion
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Cardiomyocyte tetrahydrobiopterin synthesis regulates fatty acid metabolism and susceptibility to ischaemia-reperfusion injury
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Reduced levels of tetrahydrobiopterin in diabetic mouse model: Effects on endothelial function and nitric oxide (NO) synthase dimerisation
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Cardiomyocyte tetrahydrobiopterin synthesis regulates fatty acid metabolism and susceptibility to ischaemia–reperfusion injury
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Preconditioning With Tetrahydrobiopterin Improves Outcome After Spinal Cord Ischaemia in a Small Animal Model
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Donor risk factors, retrieval technique, preservation and ischemia/reperfusion injury in pancreas transplantation.
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Tetrahydrobiopterin-dependent eNOS coupling determines vascular remodeling through accelerated endothelial regeneration
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Tetrahydrobiopterin Prevents Lung Ischemia/reperfusion-Induced Uncoupling of Endothelial Nitric Oxide Synthase
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Almmustuhtton: (2012)

Quantitative regulation of intracellular endothelial nitric-oxide synthase (eNOS) coupling by both tetrahydrobiopterin-eNOS stoichiometry and biopterin redox status: insights from cells with tet-regulated GTP cyclohydrolase I expression.
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Regulation of endothelial nitric oxide synthase (eNOS) by tetrahydrobiopterin in vascular disease
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Tetrahydrobiopterin-dependent eNOS coupling attenuates vein graft atherosclerosis through inhibition of MCP-1 mediated monocyte recruitment to sites of vascular injury
Dahkki: Ali, Z, et al.
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A requirement for Gch1 and tetrahydrobiopterin in embryonic development.
Dahkki: Douglas, G, et al.
Almmustuhtton: (2015)

Susceptibility to cardiac ischemia/reperfusion injury is modulated by chronic estrogen status.
Dahkki: Beer, S, et al.
Almmustuhtton: (2002)

Orexin A protects against cerebral ischemia-reperfusion injury by enhancing reperfusion in ischemic cortex via HIF-1α-ET-1/eNOS pathway
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Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
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Almmustuhtton: (2004)

Ratio of 5,6,7,8-tetrahydrobiopterin to 7,8-dihydrobiopterin in endothelial cells determines glucose-elicited changes in NO vs. superoxide production by eNOS.
Dahkki: Crabtree, M, et al.
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Mitophagy in hepatic ischemia-reperfusion injury
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Tetrahydrobiopterin-dependent eNOS coupling mediates endothelial regeneration and attenuates vein graft atherosclerosis in ApoE-KO mice
Dahkki: Ali, Z, et al.
Almmustuhtton: (2007)

Endothelial tetrahydrobiopterin (BH4) regulates eNOS coupling in vivo: Functional effects of BH4-eNOS stoichiometry in endothelial targeted transgenic mice
Dahkki: Bendall, J, et al.
Almmustuhtton: (2005)

GTP cyclohydrolase I gene transfer augments intracellular tetrahydrobiopterin in human endothelial cells: effects on nitric oxide synthase activity, protein levels and dimerisation.
Dahkki: Cai, S, et al.
Almmustuhtton: (2002)

Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
Dahkki: Bendall, J, et al.
Almmustuhtton: (2005)

A key role for tetrahydrobiopterin-dependent endothelial NOS regulation in vascular resistance arteries: studies in endothelial cell tetrahydrobiopterin-deficient mice
Dahkki: Chuaiphichai, S, et al.
Almmustuhtton: (2017)

Low- and high- dose treatment with folic acid, equally improve endothelial function and decrease superoxide production in human vessels via tetrahydrobiopterin-mediated eNOS coupling
Dahkki: Antoniades, C, et al.
Almmustuhtton: (2007)

Perioperative prophylaxis of renal ischemia‑reperfusion injury
Dahkki: S. V. Popov, et al.
Almmustuhtton: (2025-01-01)

Inflammation and Microvasculopathy in Renal Ischemia Reperfusion Injury
Dahkki: Daniel Patschan, et al.
Almmustuhtton: (2012-01-01)

The endothelium: gatekeeper to lung ischemia-reperfusion injury
Dahkki: Huy Q. Ta, et al.
Almmustuhtton: (2024-04-01)

Identification of the target self-antigens in ischemia/reperfusion injury
Dahkki: Zhang, M, et al.
Almmustuhtton: (2005)

Identification of the target self-antigens in ischemia/reperfusion injury.
Dahkki: Zhang, M, et al.
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Pathophysiology of Myocardial Ischemia Reperfusion Injury: A Review
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Mast cell tetrahydrobiopterin contributes to itch in mice
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Different dose-dependent effects of ebselen in sciatic nerve ischemia-reperfusion injury in rats
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