RANKL plays an important role as activator of osteoclastic bone resorption in acute Charcot osteoarthropathy

RANKL plays an important role as activator of osteoclastic bone resorption in acute Charcot osteoarthropathy

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Petrova, N, Mabilleau, G, Edmonds, M, Sabokbar, A
Μορφή:	Conference item
Έκδοση:	2007

Παρόμοια τεκμήρια

Hyperactivity of osteoclast precursors in acute charcot osteoarthropathy
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Increased osteoclastic activity in acute Charcot's osteoarthropathy: the role of receptor activator of nuclear factor-kappaB ligand
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Increased osteoclastic activity in acute Charcot's osteoarthropathy: the role of receptor activator of nuclear factor-kappaB ligand.
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Increased percentage of CD14+monocytes in acute Charcot osteoarthropathy
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Inhibition of TNF-α Reverses the Pathological Resorption Pit Profile of Osteoclasts from Patients with Acute Charcot Osteoarthropathy
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The number of CD14+osteoclast precursors in acute charcot patients is correlated to the serum levels of tnf-alpha
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Rankl-independent induction of human osteoclast formation and bone resorption by TGF-beta
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IL-6 induces human osteoclast formation and bone resorption independent of RANK/RANKL interaction
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Human osteoclast formation and bone resorption by a mechanism independent of RANK/RANKL: Role of IL-6 and IL-11.
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Number of circulating CD14-positive cells and the serum levels of TNF-α are raised in acute charcot foot.
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Bone Defect Replacement in Diabetic Neuropathic Osteoarthropathy (Charcot Foot): Review
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Interleukin-32 promotes osteoclast differentiation but not osteoclast activation
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Interleukin-32 promotes osteoclast differentiation but not osteoclast activation.
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Interleukin-32 promotes osteoclast differentiation but not osteoclast activation.
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DCIR suppresses osteoclastic proliferation and resorption by downregulating M-CSF and RANKL signaling
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The effect of particle size and electrical charge on macrophage-osteoclast differentiation and bone resorption.
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RANKL-dependent and RANKL-independent mechanisms of macrophage-osteoclast differentiation in breast cancer.
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Osteoclast precursors at sites of pathological bone resorption are CD14+mononuclear phagocytes
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M-CSF potently augments RANKL-induced resorption activation in mature human osteoclasts.
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