LOCAL ADIPONECTIN SYNTHESIS IN ADIPOSE TISSUE REGULATES VASCULAR SUPEROXIDE GENERATION IN PATIENTS WITH CORONARY ATHEROSCLEROSIS: INSIGHTS BY USING FUNCTIONAL GENETIC POLYMORPHISMS OF ADIPONECTIN GENE

Chi tiết về thư mục
Những tác giả chính:	Antoniades, C, Antonopoulos, A, Tousoulis, D, Bakogiannis, C, Miliou, A, Sfyras, N, Demosthenous, M, Psarros, C, Marinou, K, Channon, K, Stefanadis, C
Định dạng:	Conference item
Được phát hành:	2011

Interrelationships between vascular superoxide and adiponectin biosynthesis in perivascular adipose tissue in human atherosclerosis
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Genetic Variability of Adiponectin Gene Defines the Biosynthesis Rate of Adiponectin in Adipose Tissue and Regulates Superoxide Generation in Human Arteries in vivo
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Is vascular superoxide triggering adiponectin biosynthesis in perivascular adipose tissue in patients with atherosclerosis?
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Serum adiponectin regulates vascular O2-generation and NO bioavailability in patients with coronary atherosclerosis
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Associations Between Serum Adiponectin, Vascular O2-Generation and NO Bioavailability In Patients With Coronary Atherosclerosis
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Adiponectin as a Biomarker at Different Stages of Ischaemic Heart Disease: From Obesity to Ischaemic Heart Failure
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Statin treatment improves vascular redox state through a tetrahydrobiopterin-mediated re-coupling of endothelial nitric oxide synthase, in human atherosclerosis
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Novel therapeutic strategies targeting vascular redox in human atherosclerosis.
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Atorvastatin directly improves vascular redox state by affecting eNOS coupling and NADPH-oxidase activity in human vein grafts ex-vivo
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Adiponectin: from obesity to cardiovascular disease.
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Adiponectin produced in perivascular adipose tissue as a defence mechanism against vascular oxidative stress in human vein grafts
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Genetic variability on adiponectin gene affects myocardial infarction risk: the role of endothelial dysfunction.
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Genetic variability on adiponectin gene affects myocardial infarction risk: The role of endothelial dysfunction
Bằng: Antonopoulos, A, et al.
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Adiponectin: From obesity to cardiovascular disease: Etiology and Pathophysiology
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Differential responses of distinct adipose tissue depots to acute and chronic inflammation: novel insights into the complex mechanisms regulating adiponectin biosynthesis in humans
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Atorvastatin rapidly improves redox state in human vein grafts ex-vivo, by directly reducing NADPH-oxidase activity and improving eNOS coupling
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Interactions between vascular wall and perivascular adipose tissue reveal novel roles for adiponectin in the regulation of endothelial nitric oxide synthase function in human vessels.
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Reciprocal effects of systemic inflammation and brain natriuretic peptide on adiponectin biosynthesis in adipose tissue of patients with ischemic heart disease
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Reciprocal effects of systemic inflammation and brain natriuretic peptide on adiponectin biosynthesis in adipose tissue of patients with ischemic heart disease.
Bằng: Antonopoulos, A, et al.
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NADPH-oxidase activity in peripheral blood mononuclear cells as a novel inflammatory marker in atherosclerosis
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Oral Atorvastatin Treatment Improves NO Bioavailability and Vascular Redox State in the Human Arterial Wall, via a Tetrahydrobiopterin-Mediated Improvement of eNOS Coupling
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Preoperative atorvastatin treatment in CABG patients rapidly improves vein graft redox state by inhibition of Rac1 and NADPH-oxidase activity.
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Atorvastatin rapidly modifies vascular redox state and improves nitric oxide bioavailability in human vessels obtained from patients with advanced atherosclerosis
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Systemic Lipid Peroxydation and Myocardial Superoxide/Peroxynitrite Anions as Predictors of Post-Operative Outcome in Cardiac Surgery
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Rapid Effects of Atorvastatin on Tetrahydrobiopterin-Mediated eNOS Coupling in Human Arteries, is Mediated by Stimulation of Vascular GTP-Cyclohydrolase
Bằng: Antoniades, C, et al.
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Differential Effects of Acute and Chronic Inflammation on the Release of Adiponectin By Human Adipose Tissue
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Short-Term Atorvastatin Treatment Reduces Vascular Oxidative Stress Saphenous Vein Grafts by Improving eNOS Coupling and Reducing NADPH-Oxidase Activity in Patients Undergoing Elective CABG
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Adiponectin As A Link Between Type 2 Diabetes Mellitus And Vascular NADPH-Oxidase Activity In The Human Arterial Wall: The Regulatory Role Of Perivascular Adipose Tissue.
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The role of adiponectin in human vascular physiology
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The role of adiponectin in human vascular physiology.
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Nanoparticles: a promising therapeutic approach in atherosclerosis.
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Adiponectin as a regulator of vascular redox state: therapeutic implications.
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Preoperative atorvastatin treatment rapidly improves vein graft redox state by inhibiting rac1 and NADPH oxidase activity in patients undergoing elective CABG
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Homocysteine and coronary atherosclerosis: from folate fortification to the recent clinical trials.
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Mutual Regulation of Epicardial Adipose Tissue and Myocardial Redox State by PPAR-γ/Adiponectin Signalling
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Perivascular adipose tissue supresses superoxide production in internal mammary artery grafts by regulating Rac1-mediated activation of vascular NADPH-oxidase
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Platelet activation in atherogenesis associated with low-grade inflammation.
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Atorvastatin rapidly reduces myocardial superoxide and peroxynitrite generation in human atherosclerosis, by suppressing NADPH-oxidase activity independently of LDL lowering
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Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin-mediated endothelial nitric oxide synthase coupling.
Bằng: Antoniades, C, et al.
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LOCAL ADIPONECTIN SYNTHESIS IN ADIPOSE TISSUE REGULATES VASCULAR SUPEROXIDE GENERATION IN PATIENTS WITH CORONARY ATHEROSCLEROSIS: INSIGHTS BY USING FUNCTIONAL GENETIC POLYMORPHISMS OF ADIPONECTIN GENE

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