Effects of L-leucine in 5q- syndrome and other RPS14-deficient erythroblasts.

Effects of L-leucine in 5q- syndrome and other RPS14-deficient erythroblasts.

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Bibliografiset tiedot
Päätekijät:	Yip, B, Pellagatti, A, Vuppusetty, C, Giagounidis, A, Germing, U, Lamikanra, A, Roberts, D, Fernandez-Mercado, M, McDonald, E, Killick, S, Wainscoat, J, Boultwood, J
Aineistotyyppi:	Journal article
Kieli:	English
Julkaistu:	2012

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Effects of L-leucine in 5q-syndrome and other RPS14-deficient erythroblasts
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L-leucine increases translation of RPS14 and LARP1 in erythroblasts from del(5q) myelodysplastic syndrome patients
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Activation of the mTOR pathway by the amino acid (L)-leucine in the 5q- syndrome and other ribosomopathies.
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Haploinsufficiency of RPS14 in 5q- syndrome is associated with deregulation of ribosomal- and translation-related genes.
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Haploinsufficiency of RPS14 in 5q- syndrome is associated with deregulation of ribosomal- and translation-related genes (British Journal of Haematology (2008) 142, (57-64))
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Expression profiling of CD34+cells in patients with myelodysplastic syndromes with and without a del(5q)
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GENE COMPARISON STUDIES OF ERYTHROBLASTS DERIVED FROM DIFFERENT HAEMATOPOIETIC SOURCES
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Advances in the 5q- syndrome.
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Haploinsufficiency of ribosomal proteins and p53 activation in anemia: Diamond-Blackfan anemia and the 5q- syndrome.
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Marked down-regulation of nucleophosmin-1 is associated with advanced del(5q) myelodysplastic syndrome.
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Marked down-regulation of nucleophosmin-1 is associated with advanced del(5q) myelodysplastic syndrome
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Gene expression profiling of CD34+ cells in patients with the 5q- syndrome.
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MMP9 inhibition increases erythropoiesis in RPS14-deficient del(5q) MDS models through suppression of TGF-β pathways
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Integrated analyses of translatome and proteome identify the rules of translation selectivity in RPS14-deficient cells
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Induction of p53 and up-regulation of the p53 pathway in the human 5q- syndrome.
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Gene expression profiling of day 7 erythroblasts from refractory anemia with ringed sideroblasts (RARS) and microarray-based identification of erythroid granulocyte-CSF (G-CSF) targets
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Whole-exome sequencing in del(5q) myelodysplastic syndromes in transformation to acute myeloid leukemia
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Whole-exome sequencing in del(5q) myelodysplastic syndromes in transformation to acute myeloid leukemia.
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RECENT ADVANCES IN THE 5Q- SYNDROME
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High-resolution genomic profiling of myelodysplasia (MDS) by microarray comparative genomic hybridization (CGH).
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