Anti-tumor necrosis factor alpha therapy of rheumatoid arthritis. Mechanism of action.

Anti-tumor necrosis factor alpha therapy of rheumatoid arthritis. Mechanism of action.

Manylion Llyfryddiaeth
Prif Awduron:	Feldmann, M, Brennan, F, Paleolog, E, Taylor, P, Maini, R
Fformat:	Journal article
Iaith:	English
Cyhoeddwyd:	1997

Eitemau Tebyg

Similarities in the mechanisms of action of pulse corticosteroids and anti-tumor necrosis factor alpha therapy in rheumatoid arthritis - Reply
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Anti-tumor necrosis factor-alpha therapy of rheumatoid arthritis.
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Deactivation of vascular endothelium by monoclonal anti-tumor necrosis factor alpha antibody in rheumatoid arthritis.
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Use of anti-tumor necrosis factor antibodies in rheumatoid arthritis.
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Anti-TNF alpha therapy is useful in rheumatoid arthritis and Crohn's disease: analysis of the mechanism of action predicts utility in other diseases.
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What is the mechanism of action of anti-tumour necrosis factor-alpha antibody in rheumatoid arthritis?
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Systemic anti-tumor necrosis factor alpha therapy in rheumatoid arthritis down-regulates synovial tumor necrosis factor alpha synthesis.
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Modulation of angiogenic vascular endothelial growth factor by tumor necrosis factor alpha and interleukin-1 in rheumatoid arthritis.
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Anti-tumour necrosis factor specific antibody (infliximab) treatment provides insights into the pathophysiology of rheumatoid arthritis.
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Selective immunosuppression of tumour necrosis factor-alpha in rheumatoid arthritis.
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Reduction of chemokine levels and leukocyte traffic to joints by tumor necrosis factor alpha blockade in patients with rheumatoid arthritis.
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Beneficial effects of tumour necrosis factor-alpha (TNF-alpha) blockade in rheumatoid arthritis (RA)
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Cytokine expression and networks in rheumatoid arthritis: rationale for anti-TNF alpha antibody therapy and its mechanism of action.
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Expression of granulocyte-macrophage colony-stimulating factor in rheumatoid arthritis: regulation by tumor necrosis factor-alpha.
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Treatment of rheumatoid arthritis with chimeric monoclonal antibodies to tumor necrosis factor alpha.
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Treatment of rheumatoid arthritis with chimeric monoclonal antibodies to tumor necrosis factor alpha.
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A comparative study into the mechanisms of action of anti-tumor necrosis factor alpha, anti-CD4, and combined anti-tumor necrosis factor alpha/anti-CD4 treatment in early collagen-induced arthritis.
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Insights into the pathogenesis of rheumatoid arthritis from application of anti-TNF therapy.
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Similar effects of pulse corticosteroid and tumor necrosis factor a blockade in rheumatoid arthritis: Reply
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Therapeutic actions of cyclosporine and anti-tumor necrosis factor alpha in collagen-induced arthritis and the effect of combination therapy.
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Targeting TNF alpha for the therapy of rheumatoid arthritis.
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Localization of tumor necrosis factor alpha in synovial tissues and at the cartilage-pannus junction in patients with rheumatoid arthritis.
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TNF-alpha in rheumatoid arthritis and prospects of anti-TNF therapy.
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Anti-TNF alpha therapy of rheumatoid arthritis: what have we learned?
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TNF alpha blockade in rheumatoid arthritis: rationale, clinical outcomes and mechanisms of action.
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ANTI TNF THERAPY IN RHEUMATOID ARTHRITIS
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Reduction of serum matrix metalloproteinase 1 and matrix metalloproteinase 3 in rheumatoid arthritis patients following anti-tumour necrosis factor-alpha (cA2) therapy.
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Anti-TNFalpha therapy of rheumatoid arthritis: what can we learn about chronic disease?
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TNF alpha--a pivotal role in rheumatoid arthritis?
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Immunological intervention reveals reciprocal roles for tumor necrosis factor-alpha and interleukin-10 in rheumatoid arthritis and systemic lupus erythematosus.
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INCREASED EXPRESSION OF TUMOR-NECROSIS-FACTOR RECEPTORS ON CELLS ISOLATED FROM RHEUMATOID-ARTHRITIS SYNOVIAL JOINTS
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The role of TNF alpha and IL-1 in rheumatoid arthritis.
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Decrease in cellularity and expression of adhesion molecules by anti-tumor necrosis factor alpha monoclonal antibody treatment in patients with rheumatoid arthritis.
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TNF alpha is an effective therapeutic target for rheumatoid arthritis.
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