A search for polymorphism(s) in the far upstream region of the human PRO alpha 2(I) collagen gene.

A search for polymorphism(s) in the far upstream region of the human PRO alpha 2(I) collagen gene.

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Fonseca, C, Rabe, C, Bou-Gharios, G, de Crombrugghe, B, Black, C, Abraham, D, Denton, C
التنسيق:	Journal article
منشور في:	2000

مواد مشابهة

Characterization of an evolutionarily conserved far-upstream enhancer in the human alpha 2(I) collagen (COL1A2) gene.
حسب: Antoniv, T, وآخرون
منشور في: (2001)

A novel TGF-beta response element located in the human collagen type I alpha 2 far upstream enhancer is SMAD-independent
حسب: Ponticos, M, وآخرون
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Identification of the key regions within the mouse pro-alpha 2(I) collagen gene far-upstream enhancer.
حسب: De Val, S, وآخرون
منشور في: (2002)

Separate cis-acting elements in the far upstream enhancer of the mouse pro alpha 2(I) collagen (col 1 alpha 2) gene drive tissue specific expression.
حسب: Ponticos, M, وآخرون
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Activation of a fibroblast-specific enhancer of the proalpha2(I) collagen gene in tight-skin mice.
حسب: Denton, C, وآخرون
منشور في: (2001)

Cooperativity between far upstream enhancer and proximal promoter elements of the human {alpha}2(I) collagen (COL1A2) gene instructs tissue specificity in transgenic mice.
حسب: Tanaka, S, وآخرون
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Transcriptional regulation of the human alpha2(I) collagen gene
حسب: Antoniv, T, وآخرون
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Regulation of collagen type I in vascular smooth muscle cells by competition between Nkx2.5 and deltaEF1/ZEB1.
حسب: Ponticos, M, وآخرون
منشور في: (2004)

Col1a2 enhancer regulates collagen activity during development and in adult tissue repair.
حسب: Ponticos, M, وآخرون
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CTGF Contributes to collagen type I expression in the bleomycin model of lung fibrosis and in human pulmonary fibroblasts by MAPK dependent transcriptional activation
حسب: Porticos, M, وآخرون
منشور في: (2009)

CTGF overexpressing mice: a model for human scleroderma
حسب: Sonnylal, S, وآخرون
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Organ-specific collagen expression: implications for renal disease.
حسب: Alexakis, C, وآخرون
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Transcriptional regulation of the human alpha2(I) collagen gene (COL1A2), an informative model system to study fibrotic diseases.
حسب: Ramirez, F, وآخرون
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Implication of the satellite cell in dystrophic muscle fibrosis: a self-perpetuating mechanism of collagen overproduction.
حسب: Alexakis, C, وآخرون
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Bosentan alleviates the pro-fibrotic phenotype of lung scleroderma fibroblasts: Implications for treatment of scleroderma.
حسب: Leask, A, وآخرون
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Polymorphism upstream of estrogen receptor alpha reverses negative regulation of transcription.
حسب: Figtree, G, وآخرون
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Endogenous endothelin-1 signaling contributes to type I collagen and CCN2 overexpression in fibrotic fibroblasts.
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Transplantation of human fetal mesenchymal stem cells improves glomerulopathy in a collagen type I alpha 2-deficient mouse.
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Fibroblast-directed CTGF over-expression in vivo promotes systemic connective tissue fibrosis reminiscent of fibroproliferative diseases
حسب: Sonnylal, S, وآخرون
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Pivotal role of connective tissue growth factor in lung fibrosis: MAPK-dependent transcriptional activation of type I collagen.
حسب: Ponticos, M, وآخرون
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Repression of collagen type I by TGF-beta induced CUX1
حسب: Fragiadaki, M, وآخرون
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A Functional Single-Nucleotide Polymorphism Upstream of the Collagen Type III Gene Is Associated with Catastrophic Fracture Risk in Thoroughbred Horses
حسب: Esther Palomino Lago, وآخرون
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Fibroblast matrix gene expression and connective tissue remodeling: role of endothelin-1.
حسب: Shi-Wen, X, وآخرون
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Identification of a repressor in the first intron of the human alpha2(I) collagen gene (COL1A2).
حسب: Antoniv, T, وآخرون
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A commonly occurring polymorphism upstream of the estrogen receptor alpha alters transcription and is associated with increased HDL.
حسب: Figtree, G, وآخرون
منشور في: (2008)

High doses of TGF-β potently suppress type I collagen via the transcription factor CUX1.
حسب: Fragiadaki, M, وآخرون
منشور في: (2011)

Gene expression profiling of fibroblasts in response to applied bio-mechanical force
حسب: Eastwood, M, وآخرون
منشور في: (2007)

Response of human lung fibroblast cells to stimulation by endothelin-1: Involvement of ras/MEK/ERK and PI3 kinase
حسب: Shiwen, X, وآخرون
منشور في: (2003)

Non-invasive imaging for the assessment of dermal fibrosis
حسب: Jones, H, وآخرون
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Collagen type I alpha 1 gene polymorphism in premature ovarian failure
حسب: Vujović Svetlana, وآخرون
منشور في: (2013-01-01)

Endothelin is a downstream mediator of profibrotic responses to transforming growth factor beta in human lung fibroblasts.
حسب: Shi-wen, X, وآخرون
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Bosentan attenuates the profibrotic gene expression profile of lung fibroblasts in scleroderma
حسب: Shiwen, X, وآخرون
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Alpha-thalassemia caused by a large (62 kb) deletion upstream of the human alpha globin gene cluster.
حسب: Hatton, C, وآخرون
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Wound-associated macrophages control collagen 1α2 transcription during the early stages of skin wound healing.
حسب: Rodero, M, وآخرون
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Platelet-derived growth factor-beta receptor activation is essential for fibroblast and pericyte recruitment during cutaneous wound healing.
حسب: Rajkumar, V, وآخرون
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GENE EXPRESSION PROFILING OF CONNECTIVE TISSUE FIBROBLASTS IN RESPONSE TO APPLIED BIO-MECHANICAL FORCE
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Bone marrow-derived cells participate in renal regeneration but make a trivial contribution to collagen I synthesis in a murine model of kidney fibrosis
حسب: Roufosse, C, وآخرون
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Bone marrow-derived haematopoietic and mesenchymal stem cells colonise kidney but fail to produce collagen I in renal fibrosis
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Endothelin-1 induces expression of matrix-associated genes in lung fibroblasts through MEK/ERK.
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In search of the Far East /
حسب: Reader's Digest Association
منشور في: (1992)