EPITOPE MAPPING OF A NOVEL B27-SPECIFIC ANTIBODY

EPITOPE MAPPING OF A NOVEL B27-SPECIFIC ANTIBODY

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Giles, J, McHugh, K, DiGleria, K, Shaw, J, Kollnberger, S, Maenaka, K, Marroquin, O, Renner, C, Bowness, P
التنسيق:	Conference item
منشور في:	2011

مواد مشابهة

HLA-B27 homodimers differ in their interactions with LILRB1, LILRB2, KIR3DL1 and KIR3DL2 compared to HLA-B27 heterotrimers
حسب: Giles, J, وآخرون
منشور في: (2010)

Factors determining the cell surface expression of a novel HLA-B27 epitope
حسب: McHugh, K, وآخرون
منشور في: (2010)

HLA-B27 homodimers and free H chains are stronger ligands for leukocyte Ig-like receptor B2 than classical HLA class I.
حسب: Giles, J, وآخرون
منشور في: (2012)

Functional characterisation of HLA-B27 in autoimmunity
حسب: Kollnberger, S, وآخرون
منشور في: (2001)

HLA-B27 FORMS HEAVY CHAIN HOMODIMERS IN VITRO AND IN VIVO THAT BIND TO CELLULAR RECEPTORS INCLUDING LILR AND KIR FAMILY MEMBERS
حسب: McHugh, K, وآخرون
منشور في: (2010)

HLA-B27 HEAVY CHAIN DIMER EXPRESSION IN SPONDYLOARTHRITIS
حسب: Rysnik, O, وآخرون
منشور في: (2012)

HD6: A NOVEL MONOCLONAL ANTIBODY THAT RECOGNISES A SUBSET OF HLA-B27 MOLECULES STRONGLY IMPLICATED IN SPONDYLOARTHRITIS DISEASE PATHOGENESIS
حسب: McHugh, K, وآخرون
منشور في: (2011)

The pathogenesis of spondyloarthritides - HLA-B-27 homodimer expression on human monocyte/macrophage cell lines
حسب: McHugh, K, وآخرون
منشور في: (2008)

EXPRESSION OF HLA-B27 HEAVY CHAIN FORMS IN ANKYLOSING SPONDYLITIS AND HLA-B27 POSITIVE CELL LINES
حسب: Rysnik, O, وآخرون
منشور في: (2012)

HLA-B-27 heavy chain homodimer formation and natural killer cell family interaction - comparison of two HLA-B-27 subtypes in vitro
حسب: Giles, J, وآخرون
منشور في: (2008)

HD6: a novel monoclonal antibody that recognises a subset of HLA-B27 molecules strongly implicated in spondyloarthritis disease pathogenesis
حسب: McHugh, K, وآخرون
منشور في: (2011)

Inhibiting HLA-B27 homodimer-driven immune cell inflammation in spondylarthritis.
حسب: Payeli, S, وآخرون
منشور في: (2012)

Expression of aberrant HLA-B27 molecules is dependent on B27 dosage and peptide supply.
حسب: McHugh, K, وآخرون
منشور في: (2014)

TARGETING MONOCYTE-EXPRESSED HLA-B27 HOMODIMERS IN ANKYLOSING SPONDYLITIS
حسب: Bowness, P, وآخرون
منشور في: (2010)

TARGETING MONOCYTE-EXPRESSED HLA-B27 HOMODIMERS IN ANKYLOSING SPONDYLITIS
حسب: Bowness, P, وآخرون
منشور في: (2010)

THE ARTHRITIS-ASSOCIATED HLA-B(star)27:05 FORMS MORE CELL SURFACE B27 DIMER AND FREE HEAVY CHAIN LIGANDS FOR KIR3DL2 AND LILRB2 IMMUNORECEPTORS THAN HLA(star)B27:09
حسب: Shaw, J, وآخرون
منشور في: (2013)

Molecular Characterisation Of The Killer Cell Immunoglobulin-Like Receptor 3DL2 Binding To Aberrant HLA-B27 Heavy Chains In Spondyloarthritis
حسب: Hatano, H, وآخرون
منشور في: (2013)

Direct visualisation of HLA-B27 forms in cell lines and Spondyloarthropathy tissues
حسب: Rysnik, O, وآخرون
منشور في: (2013)

Ankylosing spondylitis monocytes show upregulation of proteins involved in inflammation and the ubiquitin proteasome pathway.
حسب: Wright, C, وآخرون
منشور في: (2009)

TH17 T CELLS IN ANKYLOSING SPONDYLITIS ARE POLYFUNCTIONAL AND EXPRESS HIGH LEVELS OF THE CHEMOKINE RECEPTOR CCR6
حسب: Bowness, P, وآخرون
منشور في: (2009)

THE DETECTION OF A POTENTIALLY PATHOGENIC SUBSET OF HLA-B27 CONFORMERS AT THE CELL SURFACE IN A RAT MODEL OF SPA
حسب: Mchugh, K, وآخرون
منشور في: (2012)

KIR3DL2 INTERACTION WITH HLA-B27 PROMOTES THE SURVIVAL OF TH17 CELLS IN ANKYLOSING SPONDYLITIS
حسب: Kollnberger, S, وآخرون
منشور في: (2010)

HLA-B*2709, A SUBTYPE NOT ASSOCIATED WITH ANKYLOSING SPONDYLITIS, FORMS FEWER HOMODIMERS THAN HLA-B*2705
حسب: Giles, J, وآخرون
منشور في: (2010)

HLA-B27-Homodimer-Specific Antibody Modulates the Expansion of Pro-Inflammatory T-Cells in HLA-B27 Transgenic Rats.
حسب: Osiris Marroquin Belaunzaran, وآخرون
منشور في: (2015-01-01)

THE ROLE OF HLA-B27 KIR3DL2 INTERACTIONS IN THE PATHOGENESIS OF SPONDYLOARTHRITIS
حسب: Kollnberger, S, وآخرون
منشور في: (2012)

Interaction of HLA-B27 homodimers with KIR3DL1 and KIR3DL2, unlike HLA-B27 heterotrimers, is independent of the sequence of bound peptide.
حسب: Kollnberger, S, وآخرون
منشور في: (2007)

Residue Cys-67 of HLA-B27 contributes to antigen presentation and T cell recognition of HLA-B27 restricted T cell epitopes.
حسب: Appel, H, وآخرون
منشور في: (2004)

The link between HLA-B27 and SpA--new ideas on an old problem.
حسب: McHugh, K, وآخرون
منشور في: (2012)

The KIRs of HLA-B27: the role of HLA-B27 KIR interactions in infection and spondyloarthritis
حسب: Wong-Baeza, I, وآخرون
منشور في: (2011)

Data showing non-conventional HLA-B27 expression in axial joints and gut tissue from B27 transgenic rats, and in frozen and paraffin-fixed synovial SpA tissue
حسب: Rysnik, O, وآخرون
منشور في: (2016)

The role of B27 heavy chain dimer immune receptor interactions in spondyloarthritis.
حسب: Kollnberger, S, وآخرون
منشور في: (2009)

DIRECT VISUALIZATION OF HLA-B27 FORMS IN CELL LINES AND SPONDYLOARTHROPATHY TISSUES
حسب: Rysnik, O, وآخرون
منشور في: (2013)

HLA-B*2709 FORMS LESS CELL SURFACE B27 DIMER AND FREE HEAVY CHAIN LIGANDS FOR KIR3DL2 AND LILRB2 IMMUNORECEPTORS THAN ARTHRITIS-ASSOCIATED HLA-B*2705
حسب: Shaw, J, وآخرون
منشور في: (2012)

Non-conventional forms of HLA-B27 are expressed in spondyloarthritis joints and gut tissue
حسب: Rysnik, O, وآخرون
منشور في: (2016)

Molecular determinants for KIR3DL2 binding to HLA-B27 and other HLA-class I ligands
حسب: Hatano, H, وآخرون
منشور في: (2013)

Antigen Spondylitis monocytes show upregulation of proteins involved in antigen presentation
حسب: Bowness, P, وآخرون
منشور في: (2008)

The arthritis-associated HLA-B*27:05 allele forms more cell surface B27 dimer and free heavy chain ligands for KIR3DL2 than HLA-B*27:09.
حسب: Cauli, A, وآخرون
منشور في: (2013)

Data showing non-conventional HLA-B27 expression in axial joints and gut tissue from B27 transgenic rats, and in frozen and paraffin-fixed synovial SpA tissue
حسب: Oliwia Rysnik, وآخرون
منشور في: (2016-12-01)

Crystal structures and KIR3DL1 recognition of three immunodominant viral peptides complexed to HLA-B*2705.
حسب: Stewart-Jones, G, وآخرون
منشور في: (2005)

KIR3DL2 Binds to HLA-B27 Dimers and Free H Chains More Strongly than Other HLA Class I and Promotes the Expansion of T Cells in Ankylosing Spondylitis.
حسب: Wong-Baeza, I, وآخرون
منشور في: (2013)