High energy phosphotransfer in the failing mouse heart-role of adenylate kinase and glycolytic enzymes

High energy phosphotransfer in the failing mouse heart-role of adenylate kinase and glycolytic enzymes

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Bibliografiske detaljer
Main Authors:	Aksentijevic, D, Lygate, C, Makinen, K, Sebag-Montefiore, L, Medway, D, Schneider, J, Neubauer, S
Format:	Conference item
Udgivet:	2010

Lignende værker

High-energy phosphotransfer in the failing mouse heart: role of adenylate kinase and glycolytic enzymes.
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Udgivet: (2010)

COMPENSATION FOR IMPAIRED MYOCARDIAL PHOSPHOTRANSFER IN GUANIDINOACETATE-N-METHYLTRANSFERASE KNOCKOUT MICE
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Ribose supplementation alone or with elevated creatine does not preserve high energy nucleotides or cardiac function in the failing mouse heart
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The creatine kinase energy transport system in the failing mouse heart
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Udgivet: (2006)

The creatine kinase energy transport system in the failing mouse heart.
af: Lygate, C, et al.
Udgivet: (2007)

Ribose Supplementation Alone or with Elevated Creatine Does Not Preserve High Energy Nucleotides or Cardiac Function in the Failing Mouse Heart.
af: Kiterie M E Faller, et al.
Udgivet: (2013-01-01)

Chronic creatine kinase deficiency eventually leads to congestive heart failure, but severity is dependent on genetic background, gender and age
af: Lygate, C, et al.
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Chronic creatine kinase deficiency eventually leads to congestive heart failure, but severity is dependent on genetic background, gender and age.
af: Lygate, C, et al.
Udgivet: (2012)

Metabolic Inflexibility of Malonyl CoA Decarboxylase (MCD) Knockout Mice Leads to Cardiac Remodelling and High Mortality During Peri-Weaning Period
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GUANIDINOACETATE N-METHYLTRANSFERASE KNOCKOUT MICE EXHIBIT NORMAL LEFT VENTRICULAR REMODELLING, HAEMODYNAMICS AND SURVIVAL AFTER MYOCARDIAL INFARCTION DESPITE LACK OF PHOSPHOCREATINE
af: Lygate, C, et al.
Udgivet: (2010)

Creatine uptake in mouse hearts with genetically altered creatine levels.
af: ten Hove, M, et al.
Udgivet: (2008)

Mice over-expressing the myocardial creatine transporter develop progressive heart failure and show decreased glycolytic capacity.
af: Phillips, D, et al.
Udgivet: (2010)

Cardiac phenotype of mitochondrial creatine kinase knockout mice is modified on a pure C57BL/6 genetic background.
af: Lygate, C, et al.
Udgivet: (2009)

Subtle role for adenylate kinase 1 in maintaining normal basal contractile function and metabolism in the murine heart
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Refined approach for quantification of in vivo ischemia-reperfusion injury in the mouse heart.
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Adaptations to Maintain Energy Homeostasis Compensate for Creatine-Deficiency in GAMT Knockout Mice Following Myocardial Infarction, but May Contribute to Early Mitochondrial Pathology During Aging
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Accurate infarct-size measurements from accelerated, compressed sensing reconstructed cine-MRI images in mouse hearts.
af: Wech, T, et al.
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Acetylation of muscle creatine kinase negatively impacts high-energy phosphotransfer in heart failure
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Elevated Myocardial Creatine Protects Against Ischemia/Reperfusion Injury by Improving Cardiac Energetics
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Subtle Role for Adenylate Kinase 1 in Maintaining Normal Basal Contractile Function and Metabolism in the Murine Heart
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Accelerating cine-MR imaging in mouse hearts using compressed sensing.
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In vitro Autophosphorylation and Phosphotransfer Assay of Cyanobacterial Histidine Kinase 2
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Regulation of myocardial creatine uptake capacity in genetically altered mouse models
af: Ten Hove, M, et al.
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A role for thioredoxin-interacting protein (Txnip) in cellular creatine homeostasis.
af: Zervou, S, et al.
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REGULATION OF CELLULAR CREATINE HOMEOSTASIS: A ROLE FOR THIOREDOXIN INTERACTING PROTEIN (TXNIP)
af: Zervou, S, et al.
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Moderate elevation of intracellular creatine by targeting the creatine transporter protects mice from acute myocardial infarction.
af: Lygate, C, et al.
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Phosphotransfer in Rhodobacter sphaeroides chemotaxis.
af: Porter, S, et al.
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The impact of orphan histidine kinases and phosphotransfer proteins on the regulation of clostridial sporulation initiation
af: Iman Mehdizadeh Gohari, et al.
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Accurate infarct-size measurements from accelerated, compressed sensing reconstructed cine-MRI images in mouse hearts
af: Wech Tobias, et al.
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Long-term stability of cardiac function in normal and chronically failing mouse hearts in a vertical-bore MR system.
af: Schneider, J, et al.
Udgivet: (2004)

Conformational dynamics of adenylate kinase in crystals
af: Junhyung Kim, et al.
Udgivet: (2024-01-01)

Modeling chemotaxis reveals the role of reversed phosphotransfer and a bi-functional kinase-phosphatase
af: Tindall, M, et al.
Udgivet: (2010)

Modeling chemotaxis reveals the role of reversed phosphotransfer and a Bi-functional kinase-phosphatase
af: Tindall, M, et al.
Udgivet: (2010)

Modeling chemotaxis reveals the role of reversed phosphotransfer and a bi-functional kinase-phosphatase.
af: Marcus J Tindall, et al.
Udgivet: (2010-08-01)

Fast, high-resolution in vivo cine magnetic resonance imaging in normal and failing mouse hearts on a vertical 11.7 T system.
af: Schneider, J, et al.
Udgivet: (2003)

Regulation of Myocardial Creatine Uptake by Intra- and Extracellular Creatine Levels
af: Makinen, K, et al.
Udgivet: (2008)

Modeling chemotaxis reveals the role of reversed phosphotransfer & a bi-functional kinase-phosphatase
af: Tindall, M, et al.
Udgivet: (2010)

Chronic Creatine-deficiency is not the Underlying Cause of Cardiac Dysfunction in Mice Lacking the Creatine Biosynthetic Enzyme Arginine: glycine Amidinotransferase
af: Lygate, C, et al.
Udgivet: (2013)

Unchanged Mitochondrial Organization and Compartmentation in Creatine Deficient GAMT-/- Mouse Heart
af: Branovets, J, et al.
Udgivet: (2013)

Myocardial creatine levels do not influence response to acute oxidative stress in isolated perfused heart
af: Aksentijević, D, et al.
Udgivet: (2014)