Vascular Homocysteine Versus 5-Methyl-Tetrahydrofolate As Regulators of Vascular Redox and Endothelial Function in Human Vessels

Bibliographic Details
Main Authors:	Antoniades, C, Shirodaria, C, Baarholm, O, Leeson, P, Van-Assche, T, Cunnington, C, Tousoulis, D, Stefanadis, C, Refsum, H, Channon, K
Format:	Conference item
Published:	2009

Vascular Homocysteine vs 5-Methyl-Tetrahydrofolate as Regulators of Vascular Redox and Endothelial Function in Human Vessels
by: Antoniades, C, et al.
Published: (2008)

MTHFR 677 C>T Polymorphism reveals functional importance for 5-methyltetrahydrofolate, not homocysteine, in regulation of vascular redox state and endothelial function in human atherosclerosis.
by: Antoniades, C, et al.
Published: (2009)

Plasma levels of sCD40-ligand interact with homocysteine biosynthetic pathway and predict eNOS coupling in human vessels: effects on vascular redox state
by: Antoniades, C, et al.
Published: (2008)

Genetic Polymorphism On Methyl-Tetrahydrofolate-Reductase Affects Vascular Superoxide Generation and Nitric Oxide Bioavailability By Regulating eNOS Coupling in Human Arteries and Veins
by: Antoniades, C, et al.
Published: (2009)

Soluble CD40-ligand levels are associated with vascular superoxide production and eNOS coupling in human vessels: Interrelationships with homocysteine biosynthetic pathway
by: Antoniades, C, et al.
Published: (2008)

Effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
by: Antoniades, C, et al.
Published: (2006)

Genetic variability of MTHFR affects vascular redox and nitric oxide bioavailability in human vessels, by modifying eNOS coupling
by: Antoniades, C, et al.
Published: (2008)

Genetic Variability of Methyl-Tetrahydrofolate-Reductase Modifies eNOS Coupling in Human Arteries and Veins: Effects on Vascular Superoxide Generation and Nitric Oxide Bioavailabllity
by: Antoniades, C, et al.
Published: (2008)

Rapid effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
by: Antoniades, C, et al.
Published: (2006)

Plasma asymmetrical-dimethyl-arginine (ADMA) as a regulator of vascular redox and nitric oxide bioavailability in human vessels
by: Antoniades, C, et al.
Published: (2007)

Direct effects of 5-methyl-tetrahydrofolate on vascular endotheliurn and superoxide production in human arteries and veins
by: Antoniades, C, et al.
Published: (2006)

A Novel Haplotype on GCH1 Gene, Encoding GTP Cyclohydrolase 1, Regulates Vascular Biopterin Synthesis, eNOS Coupling and Vascular Redox in Human Vessels
by: Antoniades, C, et al.
Published: (2008)

5-methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production in vessels from patients with coronary artery disease
by: Antoniades, C, et al.
Published: (2006)

GCH1 haplotype determines vascular and plasma biopterin availability in coronary artery disease effects on vascular superoxide production and endothelial function.
by: Antoniades, C, et al.
Published: (2008)

Association of plasma asymmetrical dimethylarginine (ADMA) with elevated vascular superoxide production and endothelial nitric oxide synthase uncoupling: implications for endothelial function in human atherosclerosis.
by: Antoniades, C, et al.
Published: (2009)

Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling
by: Antoniades, C, et al.
Published: (2008)

Pre-operative plasma sCD40L, endothelial function and vascular redox state predict the development of atrial fibrillation
by: Antoniades, C, et al.
Published: (2008)

5-Methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production by improving eNOS coupling and increasing tetrahydrobiopterin bioavailability in human vessels in vivo
by: Antoniades, C, et al.
Published: (2006)

5-methyl-tetrahydrofolate increases eNOS activity by preventing peroxynitrite-induced oxidation of tetrahydrobiopterin in human vessels
by: Antoniades, C, et al.
Published: (2006)

Altered plasma versus vascular biopterins in human atherosclerosis reveal relationships between endothelial nitric oxide synthase coupling, endothelial function, and inflammation.
by: Antoniades, C, et al.
Published: (2007)

The common genetic polymorphism C677T of methyltetrahydrofolate reductase regulates nitric oxide bioavailability and vascular superoxide production via modulation of eNOS coupling in human vessels
by: Antoniades, C, et al.
Published: (2007)

5-methyltetrahydrofolate rapidly improves endothelial function and decreases superoxide production in human vessels: effects on vascular tetrahydrobiopterin availability and endothelial nitric oxide synthase coupling.
by: Antoniades, C, et al.
Published: (2006)

Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels
by: Antoniades, C, et al.
Published: (2007)

Intravenous infusion of 5-methyl-tetrahydrofolate rapidly improves eNOS Coupling. In human vessels by increasing tetrahydrobiopterin bioavailability
by: Antoniades, C, et al.
Published: (2006)

Pre-Operative Estimation of Endothelial Function and Plasma sCD40L as Well as Vascular Redox State, Predict the Development of Atrial Fibrillation Post-Coronary Artery Bypass Grafting Operation
by: Antoniades, C, et al.
Published: (2008)

Relationships between vascular/plasma biopterins and endothelial nitric oxide synthase coupling, endothelial function and inflammation, in human atherosclerosis
by: Antoniades, C, et al.
Published: (2008)

Altered plasma vs. vascular biopterins in human atherosclerosis reveal relationships between endothelial nitric oxide synthase coupling, endothelial function and inflammation
by: Antomades, C, et al.
Published: (2007)

Novel therapeutic strategies targeting vascular redox in human atherosclerosis.
by: Antonopoulos, A, et al.
Published: (2009)

5-methyl-tetrahydrofolic acid increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling in human vessels in vivo: Effects on intracellular tetrahydrobiopterin oxidation
by: Antoniades, C, et al.
Published: (2006)

Atorvastatin rapidly modifies vascular redox state and improves nitric oxide bioavailability in human vessels obtained from patients with advanced atherosclerosis
by: Demosthenous, M, et al.
Published: (2010)

Homocysteine lowering: any use in atherosclerosis?
by: Antoniades, C, et al.
Published: (2007)

Tetrahydrobiopterin: a vascular redox target to improve endothelial function.
by: Channon, K
Published: (2012)

5-Methyl-tetrahydrofolic acid rapidly improves endothelial function and superoxide production in human atherosclerosis: Effects on tetrahyd robiopterin-dependent eNOS coupling
by: Antoniades, C, et al.
Published: (2006)

5-methyl-tetrahydrofolate increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling and preventing the intracellular oxidation of tetrahy-drobiopterin, in human vessels in vivo
by: Antoniades, C, et al.
Published: (2006)

Preoperative Circulating sCD40L And Vascular Redox State in LIMA Grafts, Predict The Development Of Atrial Fibrillation Post-Coronary Artery Bypass Grafting
by: Antoniades, C, et al.
Published: (2009)

Plasma asymmetrical-dimethyl-arginine affects eNOS vascular superoxide radical production and nitric oxide bioavailability in human arteries and veins
by: Antoniades, C, et al.
Published: (2008)

Statin treatment improves vascular redox state through a tetrahydrobiopterin-mediated re-coupling of endothelial nitric oxide synthase, in human atherosclerosis
by: Antoniades, C, et al.
Published: (2011)

Impaired vascular redox signalling in the vascular complications of obesity and diabetes mellitus
by: Akoumianakis, I, et al.
Published: (2017)

Progenitor cells and vascular endothelium
by: Briasoulis, A, et al.
Published: (2011)

Smoking in Asians: it doesn't stop at vascular endothelium.
by: Antoniades, C, et al.
Published: (2008)

Vascular Homocysteine Versus 5-Methyl-Tetrahydrofolate As Regulators of Vascular Redox and Endothelial Function in Human Vessels

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