Over-expression of mitochondrial creatine kinase in the murine heart improves functional recovery and protects against injury following ischaemia-reperfusion

Over-expression of mitochondrial creatine kinase in the murine heart improves functional recovery and protects against injury following ischaemia-reperfusion

Mitochondrial creatine kinase (MtCK) couples ATP production via oxidative phosphorylation to phosphocreatine in the cytosol, which acts as a mobile energy store available for regeneration of ATP at times of high demand. We hypothesised that elevating MtCK would be beneficial in ischaemia-reperfusion...

وصف كامل

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Whittington, H, Ostrowski, P, McAndrew, D, Cao, F, Shaw, A, Eykyn, T, Lake, H, Tyler, J, Schneider, J, Neubauer, S, Zervou, S, Lygate, C
التنسيق:	Journal article
اللغة:	English
منشور في:	Oxford University Press 2018

مواد مشابهة

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