Apoptotic microparticles generated during acute HIV-1 infection inhibit human dendritic cells via CD44

Apoptotic microparticles generated during acute HIV-1 infection inhibit human dendritic cells via CD44

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Bibliografiske detaljer
Main Authors:	Frleta, D, Ochoa, C, Kramer, H, Khan, SA, Stacey, A, Borrow, P, Kessler, B, Haynes, B, Bhardwaj, N
Format:	Journal article
Udgivet:	2012

Lignende værker

Apoptotic microparticles generated during acute HIV-1 infection inhibit human dendritic cells via CD44
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Udgivet: (2012-09-01)

HIV-1 infection-induced apoptotic microparticles inhibit human DCs via CD44.
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Microparticles and Apoptotic Cells Generated from Apoptotic T Cells Inhibit Human Dendritic Cell Cytokine Production
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Suppression of human dendritic cell function during acute HIV infection
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P10-05. Suppression of human dendritic cell function during acute HIV infection
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Apoptotic Cell-Derived CD14(+) Microparticles Promote the Phagocytic Activity of Neutrophilic Precursor Cells in the Phagocytosis of Apoptotic Cells
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Exposure to apoptotic activated CD4+ T cells induces maturation and APOBEC3G-mediated inhibition of HIV-1 infection in dendritic cells.
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Characterization of the effects of cross-linking of macrophage CD44 associated with increased phagocytosis of apoptotic PMN.
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Dendritic cell entry to lymphatic capillaries is orchestrated by CD44 and the hyaluronan glycocalyx
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Hydrogel Containing Anti-CD44-Labeled Microparticles, Guide Bone Tissue Formation in Osteochondral Defects in Rabbits
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IgM promotes the clearance of small particles and apoptotic microparticles by macrophages.
af: Michael L Litvack, et al.
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Chitosan Coated Microparticles Enhance Simvastatin Colon Targeting and Pro-Apoptotic Activity
af: Nabil A. Alhakamy, et al.
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Murine myeloid dendritic cells that phagocytose apoptotic T cells inhibit the immune response via NO.
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Blood Dendritic Cell Subsets in Early HIV Infection
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Apoptotic Cells Deliver Processed Antigen to Dendritic Cells for Cross-Presentation
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Apoptotic cells deliver processed antigen to dendritic cells for cross-presentation.
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Evidence of dysregulation of dendritic cells in primary HIV infection.
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CD44 and cancer.
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High apoptotic endothelial microparticle levels measured in asthma with elevated IgE and eosinophils
af: Yael Strulovici-Barel, et al.
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Elevation of intact and proteolytic fragments of acute phase proteins constitutes the earliest systemic antiviral response in HIV-1 infection
af: Kramer, H, et al.
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CD44s and CD44v6 expression in head and neck epithelia.
af: Brigitte Mack, et al.
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The Crosstalk of Apoptotic and Non-Apoptotic Signaling in CD95 System
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Elevation of intact and proteolytic fragments of acute phase proteins constitutes the earliest systemic antiviral response in HIV-1 infection.
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CD40/CD154 blockade inhibits dendritic cell expression of inflammatory cytokines but not costimulatory molecules.
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Dendritic cells that phagocytose apoptotic macrophages loaded with mycobacterial antigens activate CD8 T cells via cross-presentation.
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Autologous apoptotic neutrophils inhibit inflammatory cytokine secretion by human dendritic cells, but enhance Th1 responses
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Apoptotic Microparticles as Predicted Biomarkers in Patients with Chronic Heart Failure — Relevance to Inflammatory Cytokines and Outcomes
af: Alexander E Berezin, et al.
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Roles of Virion-Incorporated CD162 (PSGL-1), CD43, and CD44 in HIV-1 Infection of T Cells
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CD44 and cancer screening.
af: Fox, S, et al.
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CD45 is required for type I IFN production by dendritic cells.
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The bactericidal effect of dendritic copper microparticles, contained in an alginate matrix, on Escherichia coli.
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OA031-05. HIV escape from natural killer cytotoxicity: Nef inhibits NKp44L expression on HIV-infected CD4+ T cells
af: Debre P, et al.
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The Role of CD44 in Disease Pathophysiology And Targeted Treatment
af: Andre R. Jordan, et al.
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Innate immune responses in primary HIV-1 infection.
af: Borrow, P, et al.
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Ontogeny of CD8+T Cells in Acute HIV-1 that Inhibit Autologous and Heterologous Transmitted/Founder (T/F) Viruses
af: Freel, SA, et al.
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Post-translational modifications, subcellular relocation and release in apoptotic microparticles: apoptosis turns nuclear proteins into autoantigens.
af: Sylviane Muller, et al.
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