COMPENSATION FOR IMPAIRED MYOCARDIAL PHOSPHOTRANSFER IN GUANIDINOACETATE-N-METHYLTRANSFERASE KNOCKOUT MICE
Main Authors: | Aksentijevic, D, Lygate, C, Makinen, K, Sebag-Montefiore, L, Medway, D, Neubauer, S |
---|---|
Format: | Conference item |
Published: |
2010
|
Similar Items
-
GUANIDINOACETATE N-METHYLTRANSFERASE KNOCKOUT MICE EXHIBIT NORMAL LEFT VENTRICULAR REMODELLING, HAEMODYNAMICS AND SURVIVAL AFTER MYOCARDIAL INFARCTION DESPITE LACK OF PHOSPHOCREATINE
by: Lygate, C, et al.
Published: (2010) -
High energy phosphotransfer in the failing mouse heart-role of adenylate kinase and glycolytic enzymes
by: Aksentijevic, D, et al.
Published: (2010) -
Age-dependent decline in cardiac function in guanidinoacetate-N-methyltransferase knockout mice
by: Aksentijević, D, et al.
Published: (2019) -
High-energy phosphotransfer in the failing mouse heart: role of adenylate kinase and glycolytic enzymes.
by: Aksentijević, D, et al.
Published: (2010) -
Guanidinoacetate N-methyltransferase-knockout mice completely lacking myocardial phosphocreatine show normal in vivo cardiac function, but increased susceptibility to ischemia/reperfusion injury
by: Lygate, C, et al.
Published: (2004)