Human osteoclast formation and bone resorption by a mechanism independent of RANK/RANKL: Role of IL-6 and IL-11.

Human osteoclast formation and bone resorption by a mechanism independent of RANK/RANKL: Role of IL-6 and IL-11.

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Kudo, O, Sabokbar, A, Itonaga, I, Fujikawa, Y, Torisu, T, Athanasou, N
Μορφή:	Journal article
Έκδοση:	2001

Παρόμοια τεκμήρια

IL-6 induces human osteoclast formation and bone resorption independent of RANK/RANKL interaction
ανά: Kudo, O, κ.ά.
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Rankl-independent induction of human osteoclast formation and bone resorption by TGF-beta
ανά: Itonaga, I, κ.ά.
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Interleukin-6 and interleukin-11 support human osteoclast formation by a RANKL-independent mechanism.
ανά: Kudo, O, κ.ά.
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Proinflammatory cytokine (TNFalpha/IL-1alpha) induction of human osteoclast formation.
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Transforming growth factor-beta induces osteoclast formation in the absence of RANKL.
ανά: Itonaga, I, κ.ά.
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Cellular and humoral mechanisms of osteoclast formation and bone resorption in Gorham-Stout disease.
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Involvement of RANKL-dependent/independent mechanisms in osteoclast formation in aseptic loosening
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Bone stromal cells in pagetic bone and Paget's sarcoma express RANKL and support human osteoclast formation.
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Human osteoclast formation and bone resorption by monocytes and synovial macrophages in rheumatoid arthritis.
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Two distinct cellular mechanisms of osteoclast formation and bone resorption in periprosthetic osteolysis.
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Cellular mechanisms of osteoclast formation and bone resorption in rheumatoid arthritis
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Human osteoclast ontogeny and pathological bone resorption.
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Cellular mechanisms of osteoclast formation and lacunar resorption in giant cell granuloma of the jaw.
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The effect of macrophage-colony stimulating factor and other humoral factors (interleukin-1, -3, -6, and -11, tumor necrosis factor-alpha, and granulocyte macrophage-colony stimulating factor) on human osteoclast formation from circulating cells.
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The role of rank ligand in RA synovial macrophage-osteoclast differentiation
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RANKL-dependent and RANKL-independent mechanisms of macrophage-osteoclast differentiation in breast cancer.
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RANKL plays an important role as activator of osteoclastic bone resorption in acute Charcot osteoarthropathy
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Osteoclast precursors at sites of pathological bone resorption are CD14+mononuclear phagocytes
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The effect of particle size and electrical charge on macrophage-osteoclast differentiation and bone resorption.
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Macrophage-osteoclast differentiation and bone resorption in osteoarthrotic subchondral acetabular cysts.
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Synovial fluid macrophages are capable of osteoclast formation and resorption.
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Humoral factors act directly on circulating osteoclast precursors to control osteoclast formation
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ELMO1 Regulates RANKL-Stimulated Differentiation and Bone Resorption of Osteoclasts
ανά: Xinyue Liang, κ.ά.
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Arthroplasty membrane-derived fibroblasts directly induce osteoclast formation and osteolysis in aseptic loosening.
ανά: Sabokbar, A, κ.ά.
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Major sex and age differences in human osteoclast formation from circulating precursors: Implications for the pathogenesis of primary osteoporosis
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Macrophage colony-stimulating factor and interleukin-6 release by periprosthetic cells stimulates osteoclast formation and bone resorption.
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Light, a TNF-superfamily member, induces osteoclast formation in vitro; A novel mediator of bone resorption?
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Direct induction by melanoma, prostate cancer and Ewing's sarcoma tumour cells of osteoclast formation by a RANKL-independent mechanism
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Osteoclast formation in Gorham-Stout disease
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Suppressive effects of (-)-tubaic acid on RANKL-induced osteoclast differentiation and bone resorption
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Radio-opaque agents in bone cement increase bone resorption.
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