Endogenous nitric oxide formation in cardiac myocytes does not control respiration during β‐adrenergic stimulation

Endogenous nitric oxide formation in cardiac myocytes does not control respiration during β‐adrenergic stimulation

Endothelial nitric oxide (NO) controls cardiac oxygen (O2) consumption in a paracrine way by slowing respiration at the mitochondrial electron transport chain. While NO synthases (NOSs) are also expressed in cardiac myocytes, it is unclear whether they control respiration in an intracrine way. Furth...

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Chi tiết về thư mục
Những tác giả chính:	Kohlhaas, M, Nickel, A, Bergem, S, Casadei, B, Laufs, U, Maack, C
Định dạng:	Journal article
Ngôn ngữ:	English
Được phát hành:	Wiley 2017

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