Thiamine and benfotiamine improve cognition and ameliorate GSK-3β-associated stress-induced behaviours in mice.
Thiamine (vitamin B1) deficiency in the brain has been implicated in the development of dementia and symptoms of depression. Indirect evidence suggests that thiamine may contribute to these pathologies by controlling the activities of glycogen synthase kinase (GSK)-3β. While decreased G...
প্রধান লেখক: | Markova, N, Bazhenova, N, Anthony, D, Vignisse, J, Svistunov, A, Lesch, K, Bettendorff, L, Strekalova, T |
---|---|
বিন্যাস: | Journal article |
ভাষা: | English |
প্রকাশিত: |
Elsevier
2016
|
অনুরূপ উপাদানগুলি
অনুরূপ উপাদানগুলি
-
Thiamine and benfotiamine prevent stress-induced suppression of hippocampal neurogenesis in mice exposed to predation without affecting brain thiamine diphosphate levels
অনুযায়ী: Vignisse, J, অন্যান্য
প্রকাশিত: (2017) -
Thiamine and benfotiamine: Focus on their therapeutic potential
অনুযায়ী: Iva Bozic, অন্যান্য
প্রকাশিত: (2023-11-01) -
Chemoreactomic analysis of thiamine disulfide, thiamine hydrochloride, and benfotiamine molecules
অনুযায়ী: O. A. Gromova, অন্যান্য
প্রকাশিত: (2017-07-01) -
Benfotiamine Supplementation Increases Thiamine in Muscle of Endurance-Trained Mice and Affects the Energy Metabolism
অনুযায়ী: Álisson C. Gonçalves, অন্যান্য
প্রকাশিত: (2024-01-01) -
Prostatic acid phosphatase is required for the antinociceptive effects of thiamine and benfotiamine.
অনুযায়ী: Julie K Hurt, অন্যান্য
প্রকাশিত: (2012-01-01)