Thiamine and benfotiamine improve cognition and ameliorate GSK-3β-associated stress-induced behaviours in mice.

Thiamine and benfotiamine improve cognition and ameliorate GSK-3β-associated stress-induced behaviours in mice.

Thiamine (vitamin B1) deficiency in the brain has been implicated in the development of dementia and symptoms of depression. Indirect evidence suggests that thiamine may contribute to these pathologies by controlling the activities of glycogen synthase kinase (GSK)-3β. While decreased G...

Bibliográfalaš dieđut
Váldodahkkit:	Markova, N, Bazhenova, N, Anthony, D, Vignisse, J, Svistunov, A, Lesch, K, Bettendorff, L, Strekalova, T
Materiálatiipa:	Journal article
Giella:	English
Almmustuhtton:	Elsevier 2016

Geahča maid

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Frontal Bone Insufficiency in Gsk3β Mutant Mice.
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Novel reporter alleles of GSK-3α and GSK-3β.
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Phosphorylation of protein Tau by GSK3β prolongs survival of bigenic Tau.P301L × GSK3β mice by delaying brainstem tauopathy
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Correction: Inhibition of GSK-3 Ameliorates Aβ Pathology in an Adult-Onset Drosophila Model of Alzheimer's Disease
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Almmustuhtton: (2012-04-01)

GSK3β and the aging kidney
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Almmustuhtton: (2022-02-01)

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Almmustuhtton: (2012)

Thiamine diphosphate in whole blood, thiamine and thiamine monophosphate in breast-milk in a refugee population
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Chinese Medicine, Succinum, Ameliorates Cognitive Impairment of Carotid Artery Ligation Rats and Inhibits Apoptosis of HT22 Hippocampal Cells via Regulation of the GSK3β/β-Catenin Pathway
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Fucoidan–Fucoxanthin Ameliorated Cardiac Function via IRS1/GRB2/ SOS1, GSK3β/CREB Pathways and Metabolic Pathways in Senescent Mice
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