Cardiac cholinergic NO-cGMP signaling following acute myocardial infarction and nNOS gene transfer.

Cardiac cholinergic NO-cGMP signaling following acute myocardial infarction and nNOS gene transfer.

Myocardial infarction (MI) is associated with oxidative stress, which may cause cardiac autonomic impairment. We tested the hypothesis that acute MI disrupts cardiac cholinergic signaling by impairing nitric oxide (NO)-cGMP modulation of acetylcholine (ACh) release and whether the restoration of thi...

Fuld beskrivelse

Bibliografiske detaljer
Main Authors:	Dawson, T, Li, D, Woodward, T, Barber, Z, Wang, L, Paterson, D
Format:	Journal article
Sprog:	English
Udgivet:	2008

Lignende værker

Myocardial nNOS-derived NO regulates excitation-contraction coupling via differential and cGMP-independent effects on protein phosphorylation
af: Zhang, M, et al.
Udgivet: (2012)

Targeted nNOS gene transfer into the cardiac vagus rapidly increases parasympathetic function in the pig.
af: Heaton, D, et al.
Udgivet: (2005)

NO-cGMP pathway facilitates vagal neurotransmission and bradycardia
af: Herring, N, et al.
Udgivet: (2001)

Impaired cAMP-cGMP cross-talk during cardiac sympathetic dysautonomia.
af: Bardsley, E, et al.
Udgivet: (2016)

Cardiac nNOS regulates myocardial relaxation by stimulating phospholamban phosphorylation
af: Zhang, Y, et al.
Udgivet: (2005)

Cardiac nNOS regulates myocardial relaxation by stimulating phospholamban phosphorylation
af: Zhang, Y, et al.
Udgivet: (2005)

nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction
af: Dawson, D, et al.
Udgivet: (2005)

nNOS gene deletion exacerbates pathological left ventricular remodelling and functional deterioration after myocardial infarction
af: Dawson, D, et al.
Udgivet: (2005)

nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction.
af: Dawson, D, et al.
Udgivet: (2005)

cGMP catabolism by phosphodiesterase 5A regulates cardiac adrenergic stimulation by NOS3-dependent mechanism.
af: Takimoto, E, et al.
Udgivet: (2005)

Natriuretic peptides like NO facilitate cardiac vagal neurotransmission and bradycardia via a cGMP pathway.
af: Herring, N, et al.
Udgivet: (2001)

Adenoviral nNOS gene transfer into the cardiac vagus increases protein expression within 9 hours and enhances vagal responsiveness in the pig
af: Paterson, D, et al.
Udgivet: (2004)

NO-cGMP dependent pathway and the modulation of vagal control of heart rate
af: Herring, N, et al.
Udgivet: (2000)

Cardiac nitric oxide: emerging role for nNOS in regulating physiological function.
af: Danson, E, et al.
Udgivet: (2005)

Discordant response to exercise training in nnos knockout mice: Differential role of nNOS on exercise performance versus cardiac hypertrophy
af: de Bono, J, et al.
Udgivet: (2007)

FRET-based cyclic GMP biosensors measure low cGMP concentrations in cardiomyocytes and neurons
af: Calamera, G, et al.
Udgivet: (2019)

FRET-based cyclic GMP biosensors measure low cGMP concentrations in cardiomyocytes and neurons
af: Calamera, G, et al.
Udgivet: (2019)

Possible regulation of cardiac ADP-ribosyl cyclase by cGMP-dependent mechanisms
af: Solanki, P, et al.
Udgivet: (2000)

Sulphonylurea-sensitive channels and NO-cGMP pathway modulate the heart rate response to vagal nerve stimulation in vitro.
af: Almond, S, et al.
Udgivet: (2000)

Role of nNOS in the automatic control of cardiac excitability in cardiac physiological and pathophysiological states
af: Heaton, D, et al.
Udgivet: (2005)

Activation of sulphonylurea-sensitive channels and the NO-cGMP pathway decreases the heart rate response to sympathetic nerve stimulation.
af: Mohan, R, et al.
Udgivet: (2000)

Selective gene deletion of the neuronal nitric oxide synthase (nNOS) accelerates pathological left ventricular remodelling and functional deterioration after myocardial infarction (MI)
af: Dawson, D, et al.
Udgivet: (2005)

Effect of disruption of the nNOS gene on myocardial calcium current and contraction
af: Sears, C, et al.
Udgivet: (2001)

Nutraceutical COMP-4 confers protection against endothelial dysfunction through the eNOS/iNOS-NO-cGMP pathway.
af: Monica G Ferrini, et al.
Udgivet: (2025-01-01)

Association between left ventricular reverse remodelling and the B-type natriuretic peptide–cGMP cascade after anterior acute myocardial infarction
af: Satoshi Yasuda, et al.
Udgivet: (2025-01-01)

cAMP and cGMP signaling cross-talk: role of phosphodiesterases and implications for cardiac pathophysiology.
af: Zaccolo, M, et al.
Udgivet: (2007)

NO-cGMP pathway increases the hyperpolarisation-activated current, I(f), and heart rate during adrenergic stimulation.
af: Herring, N, et al.
Udgivet: (2001)

L-NAME causes antinociception by stimulation of the arginine-NO-cGMP pathway
af: I. D. G. Duarte, et al.
Udgivet: (2000-01-01)

Pre-synaptic NO-cGMP pathway modulates vagal control of heart rate in isolated adult guinea pig atria.
af: Herring, N, et al.
Udgivet: (2000)

Constitutive NO production in the myocardium: More than cGMP signalling
af: Casadei, B
Udgivet: (2008)

Role of the NO-GC/cGMP signaling pathway in platelet biomechanics
af: Aylin Balmes, et al.
Udgivet: (2024-12-01)

Effectiveness of NO-cGMP axis for lower urinary tract syndrome
af: Yukihide Matsui, et al.
Udgivet: (2016-06-01)

Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
af: Zhang, Y, et al.
Udgivet: (2006)

Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
af: Zhang, Y, et al.
Udgivet: (2005)

Nitric oxide-cGMP pathway facilitates acetylcholine release and bradycardia during vagal nerve stimulation in the guinea-pig in vitro.
af: Herring, N, et al.
Udgivet: (2001)

Differential role of nNOS in heart rate regulation between rest and exercise
af: Adlam, D, et al.
Udgivet: (2007)

Sodium channel and nNOS expression following inferior alveolar nerve
af: Davies, S, et al.
Udgivet: (2001)

nNOS expression following inferior alveolar nerve injury in the ferret.
af: Davies, S, et al.
Udgivet: (2004)

Compartmentalized phosphodiesterase-2 activity blunts beta-adrenergic cardiac inotropy via an NO/cGMP-dependent pathway.
af: Mongillo, M, et al.
Udgivet: (2006)

Role of cGMP-inhibited phosphodiesterase and sarcoplasmic calcium in mediating the increase in basal heart rate with nitric oxide donors.
af: Musialek, P, et al.
Udgivet: (2000)