Cardiac cholinergic NO-cGMP signaling following acute myocardial infarction and nNOS gene transfer.

Cardiac cholinergic NO-cGMP signaling following acute myocardial infarction and nNOS gene transfer.

Myocardial infarction (MI) is associated with oxidative stress, which may cause cardiac autonomic impairment. We tested the hypothesis that acute MI disrupts cardiac cholinergic signaling by impairing nitric oxide (NO)-cGMP modulation of acetylcholine (ACh) release and whether the restoration of thi...

Cur síos iomlán

Sonraí bibleagrafaíochta
Príomhchruthaitheoirí:	Dawson, T, Li, D, Woodward, T, Barber, Z, Wang, L, Paterson, D
Formáid:	Journal article
Teanga:	English
Foilsithe / Cruthaithe:	2008

Míreanna comhchosúla

Myocardial nNOS-derived NO regulates excitation-contraction coupling via differential and cGMP-independent effects on protein phosphorylation
de réir: Zhang, M, et al.
Foilsithe / Cruthaithe: (2012)

Targeted nNOS gene transfer into the cardiac vagus rapidly increases parasympathetic function in the pig.
de réir: Heaton, D, et al.
Foilsithe / Cruthaithe: (2005)

NO-cGMP pathway facilitates vagal neurotransmission and bradycardia
de réir: Herring, N, et al.
Foilsithe / Cruthaithe: (2001)

Impaired cAMP-cGMP cross-talk during cardiac sympathetic dysautonomia.
de réir: Bardsley, E, et al.
Foilsithe / Cruthaithe: (2016)

Cardiac nNOS regulates myocardial relaxation by stimulating phospholamban phosphorylation
de réir: Zhang, Y, et al.
Foilsithe / Cruthaithe: (2005)

Cardiac nNOS regulates myocardial relaxation by stimulating phospholamban phosphorylation
de réir: Zhang, Y, et al.
Foilsithe / Cruthaithe: (2005)

nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction
de réir: Dawson, D, et al.
Foilsithe / Cruthaithe: (2005)

nNOS gene deletion exacerbates pathological left ventricular remodelling and functional deterioration after myocardial infarction
de réir: Dawson, D, et al.
Foilsithe / Cruthaithe: (2005)

nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction.
de réir: Dawson, D, et al.
Foilsithe / Cruthaithe: (2005)

cGMP catabolism by phosphodiesterase 5A regulates cardiac adrenergic stimulation by NOS3-dependent mechanism.
de réir: Takimoto, E, et al.
Foilsithe / Cruthaithe: (2005)

Natriuretic peptides like NO facilitate cardiac vagal neurotransmission and bradycardia via a cGMP pathway.
de réir: Herring, N, et al.
Foilsithe / Cruthaithe: (2001)

Adenoviral nNOS gene transfer into the cardiac vagus increases protein expression within 9 hours and enhances vagal responsiveness in the pig
de réir: Paterson, D, et al.
Foilsithe / Cruthaithe: (2004)

NO-cGMP dependent pathway and the modulation of vagal control of heart rate
de réir: Herring, N, et al.
Foilsithe / Cruthaithe: (2000)

Cardiac nitric oxide: emerging role for nNOS in regulating physiological function.
de réir: Danson, E, et al.
Foilsithe / Cruthaithe: (2005)

Discordant response to exercise training in nnos knockout mice: Differential role of nNOS on exercise performance versus cardiac hypertrophy
de réir: de Bono, J, et al.
Foilsithe / Cruthaithe: (2007)

FRET-based cyclic GMP biosensors measure low cGMP concentrations in cardiomyocytes and neurons
de réir: Calamera, G, et al.
Foilsithe / Cruthaithe: (2019)

FRET-based cyclic GMP biosensors measure low cGMP concentrations in cardiomyocytes and neurons
de réir: Calamera, G, et al.
Foilsithe / Cruthaithe: (2019)

Possible regulation of cardiac ADP-ribosyl cyclase by cGMP-dependent mechanisms
de réir: Solanki, P, et al.
Foilsithe / Cruthaithe: (2000)

Sulphonylurea-sensitive channels and NO-cGMP pathway modulate the heart rate response to vagal nerve stimulation in vitro.
de réir: Almond, S, et al.
Foilsithe / Cruthaithe: (2000)

Role of nNOS in the automatic control of cardiac excitability in cardiac physiological and pathophysiological states
de réir: Heaton, D, et al.
Foilsithe / Cruthaithe: (2005)

Activation of sulphonylurea-sensitive channels and the NO-cGMP pathway decreases the heart rate response to sympathetic nerve stimulation.
de réir: Mohan, R, et al.
Foilsithe / Cruthaithe: (2000)

Selective gene deletion of the neuronal nitric oxide synthase (nNOS) accelerates pathological left ventricular remodelling and functional deterioration after myocardial infarction (MI)
de réir: Dawson, D, et al.
Foilsithe / Cruthaithe: (2005)

Effect of disruption of the nNOS gene on myocardial calcium current and contraction
de réir: Sears, C, et al.
Foilsithe / Cruthaithe: (2001)

Nutraceutical COMP-4 confers protection against endothelial dysfunction through the eNOS/iNOS-NO-cGMP pathway.
de réir: Monica G Ferrini, et al.
Foilsithe / Cruthaithe: (2025-01-01)

Association between left ventricular reverse remodelling and the B-type natriuretic peptide–cGMP cascade after anterior acute myocardial infarction
de réir: Satoshi Yasuda, et al.
Foilsithe / Cruthaithe: (2025-01-01)

cAMP and cGMP signaling cross-talk: role of phosphodiesterases and implications for cardiac pathophysiology.
de réir: Zaccolo, M, et al.
Foilsithe / Cruthaithe: (2007)

NO-cGMP pathway increases the hyperpolarisation-activated current, I(f), and heart rate during adrenergic stimulation.
de réir: Herring, N, et al.
Foilsithe / Cruthaithe: (2001)

L-NAME causes antinociception by stimulation of the arginine-NO-cGMP pathway
de réir: I. D. G. Duarte, et al.
Foilsithe / Cruthaithe: (2000-01-01)

Pre-synaptic NO-cGMP pathway modulates vagal control of heart rate in isolated adult guinea pig atria.
de réir: Herring, N, et al.
Foilsithe / Cruthaithe: (2000)

Constitutive NO production in the myocardium: More than cGMP signalling
de réir: Casadei, B
Foilsithe / Cruthaithe: (2008)

Role of the NO-GC/cGMP signaling pathway in platelet biomechanics
de réir: Aylin Balmes, et al.
Foilsithe / Cruthaithe: (2024-12-01)

Effectiveness of NO-cGMP axis for lower urinary tract syndrome
de réir: Yukihide Matsui, et al.
Foilsithe / Cruthaithe: (2016-06-01)

Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
de réir: Zhang, Y, et al.
Foilsithe / Cruthaithe: (2006)

Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
de réir: Zhang, Y, et al.
Foilsithe / Cruthaithe: (2005)

Nitric oxide-cGMP pathway facilitates acetylcholine release and bradycardia during vagal nerve stimulation in the guinea-pig in vitro.
de réir: Herring, N, et al.
Foilsithe / Cruthaithe: (2001)

Differential role of nNOS in heart rate regulation between rest and exercise
de réir: Adlam, D, et al.
Foilsithe / Cruthaithe: (2007)

Sodium channel and nNOS expression following inferior alveolar nerve
de réir: Davies, S, et al.
Foilsithe / Cruthaithe: (2001)

nNOS expression following inferior alveolar nerve injury in the ferret.
de réir: Davies, S, et al.
Foilsithe / Cruthaithe: (2004)

Compartmentalized phosphodiesterase-2 activity blunts beta-adrenergic cardiac inotropy via an NO/cGMP-dependent pathway.
de réir: Mongillo, M, et al.
Foilsithe / Cruthaithe: (2006)

Role of cGMP-inhibited phosphodiesterase and sarcoplasmic calcium in mediating the increase in basal heart rate with nitric oxide donors.
de réir: Musialek, P, et al.
Foilsithe / Cruthaithe: (2000)