Mycobacterium tuberculosis modulates miR-106b-5p to control cathepsin S expression resulting in higher pathogen survival and poor T-cell activation
The success of tuberculosis (TB) bacillus, Mycobacterium tuberculosis (Mtb), relies on the ability to survive in host cells and escape to immune surveillance and activation. We recently demonstrated that Mtb manipulation of host lysosomal cathepsins in macrophages leads to decreased enzymatic activi...
Main Authors: | Pires, D, Bernard, EM, Pombo, JP, Carmo, N, Fialho, C, Gutierrez, MG, Bettencourt, P, Anes, E |
---|---|
Format: | Journal article |
Language: | English |
Published: |
Frontiers Media
2017
|
Similar Items
-
Role of cathepsins in Mycobacterium tuberculosis survival in human macrophages
by: Pires, D, et al.
Published: (2016) -
Modulation of Cystatin F in Human Macrophages Impacts Cathepsin-Driven Killing of Multidrug-Resistant <i>Mycobacterium tuberculosis</i>
by: Manoj Mandal, et al.
Published: (2023-07-01) -
Liposomal Delivery of Saquinavir to Macrophages Overcomes Cathepsin Blockade by <i>Mycobacterium tuberculosis</i> and Helps Control the Phagosomal Replicative Niches
by: David Pires, et al.
Published: (2023-01-01) -
miR-106b as an emerging therapeutic target in cancer
by: Surendra Kumar Sagar
Published: (2022-07-01) -
Effect of taxanes on the miR-106 and miR-200c expression in prostate cancer cells in vivo and in vitro
by: D. S. Plevako, et al.
Published: (2023-01-01)