CIGMA as a tool in the study of NIDDM.

CIGMA as a tool in the study of NIDDM.

Bibliografische gegevens
Hoofdauteurs:	Turner, R, Rudenski, A, Hosker, J, Matthews, DR, Levy, J, O'Rahilly, S
Formaat:	Journal article
Taal:	English
Gepubliceerd in:	1988

Gelijkaardige items

Pathogenesis of NIDDM--a disease of deficient insulin secretion.
door: Turner, R, et al.
Gepubliceerd in: (1988)

ASSESSMENT OF B-CELL FUNCTION AND INSULIN RESISTANCE IN TYPE-II DIABETES BY CONTINUOUS INFUSION OF GLUCOSE WITH MODEL ASSESSMENT (CIGMA)
door: Turner, R, et al.
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MEASUREMENT OF INSULIN-RESISTANCE AND BETA-CELL FUNCTION - THE HOMA AND CIGMA APPROACH
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Application of structural model of glucose-insulin relations to assess beta-cell function and insulin sensitivity.
door: Turner, R, et al.
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The glucose stimulus-response curve of the beta-cell in physically trained humans, assessed by hyperglycemic clamps.
door: O'Rahilly, S, et al.
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Metabolic abnormalities in children of non-insulin dependent diabetics.
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Evaluation of insulin sensitivity: the HOMA and CIGMA models
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[Evaluation of insulin sensitivity: the HOMA and CIGMA models]
door: Levy, J
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Quantitative modelling of endocrine diseases as exemplified by diabetes.
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Does type II diabetes arise from a major gene defect producing insulin resistance or beta cell dysfunction?
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Diabetes nomenclature: classification or grading of severity?
door: Turner, R, et al.
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Segregation analysis of NIDDM in Caucasian families.
door: Cook, J, et al.
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ESTIMATION OF INSULIN-SECRETION RATE BY DECONVOLUTION ANALYSIS OF C-PEPTIDE AND OF INSULIN GIVES CONSISTENT RESULTS
door: Rudenski, A, et al.
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The beta cell glucose stimulus-response curve in normal humans assessed by insulin and C-peptide secretion rates.
door: Rudenski, A, et al.
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Similar reduction of first- and second-phase B-cell responses at three different glucose levels in type II diabetes and the effect of gliclazide therapy.
door: Hosker, J, et al.
Gepubliceerd in: (1989)

Impaired pulsatile secretion of insulin in relatives of patients with non-insulin-dependent diabetes.
door: O'Rahilly, S, et al.
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No glycemic benefit from guar administration in NIDDM.
door: Holman, R, et al.
Gepubliceerd in: (1987)

Relative hyperproinsulinemia of NIDDM persists despite the reduction of hyperglycemia with insulin or sulfonylurea therapy.
door: Rachman, J, et al.
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Homeostasis model assessment: insulin resistance and beta-cell function from fasting plasma glucose and insulin concentrations in man.
door: Matthews, DR, et al.
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Post-prandial amylin concentrations are increased by sulphonylurea, but unchanged by basal insulin, in NIDDM
door: Rachman, J, et al.
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Insulin use in NIDDM. Rationale based on pathophysiology of disease.
door: Turner, R, et al.
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Risk factors predicting progression of diabetic retinopathy over six years in 1579 NIDDM patients
door: Stratton, I, et al.
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Leucine is a better priming agent than arginine for insulin secretion in gliclazide treated NIDDM
door: Matthews, DR, et al.
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RESTING THE BETA-CELLS WITH INSULIN IN NIDDM DOES NOT IMPROVE BETA-CELL FUNCTION NOR PROINSULIN PROCESSING
door: Rachman, J, et al.
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No evidence for excess allele-sharing on chromosome 2q (region of NIDDM1) in a large sibship collection
door: Cassell, P, et al.
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Familial association and lambda s for diabetes, obesity, high triglycerides and low HDL-cholesterol in sibs of NIDDM patients
door: Levy, J, et al.
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Familial association and lambda s for diabetes, obesity, high triglycerides and low HDL-cholesterol in sibs of NIDDM patients.
door: Levy, J, et al.
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Treatment of NIDDM - Progressive requirement for polypharmacy to attain glycaemic goals
door: Cull, C, et al.
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LOSS OF NORMAL SIGMOID, BETA-CELL DOSE-RESPONSE CURVE TO GLUCOSE IN NON-INSULIN-DEPENDENT DIABETES
door: Hosker, J, et al.
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Near-normalisation of diurnal glucose concentrations by continuous administration of glucagon-like peptide-1 (GLP-1) in subjects with NIDDM.
door: Rachman, J, et al.
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Continuous infusion of glucose with model assessment: measurement of insulin resistance and beta-cell function in man.
door: Hosker, J, et al.
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Changes in amylin and amylin-like peptide concentrations and beta-cell function in response to sulfonylurea or insulin therapy in NIDDM.
door: Rachman, J, et al.
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An appreciation of Robert Turner.
door: O'Rahilly, S, et al.
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Effects of ethnicity on metabolic control during the first six years of NIDDM
door: Davis, T, et al.
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Normalization of insulin responses to glucose by overnight infusion of glucagon-like peptide 1 (7-36) amide in patients with NIDDM.
door: Rachman, J, et al.
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A STUDY OF MOXONIDINE’S METABOLIC EFFECTS IN PATIENTS WITH ARTERIAL HYPERTENSION AND NIDDM
door: D. V. Nebieridze, et al.
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Unsteady Natural Convection in a Cylindrical Containment Vessel (CIGMA) With External Wall Cooling: Numerical CFD Simulation
door: Ari Hamdani, et al.
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UK prospective diabetes study (UKPDS) 14: association of angiotensin-converting enzyme insertion/deletion polymorphism with myocardial infarction in NIDDM.
door: Keavney, B, et al.
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Coronary heart disease and risk factors in NIDDM--experience from the United Kingdom Prospective Diabetes Study.
door: Turner, R, et al.
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Understanding "insulin resistance": both glucose resistance and insulin resistance are required to model human diabetes.
door: Rudenski, A, et al.
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