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Impaired exercise capacity in...
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Impaired exercise capacity in cytosolic (M-CK) but not in mitochondrial creatine kinase knockout mice
Manylion Llyfryddiaeth
Prif Awduron:
Maass, A
,
Bauer, L
,
Neubauer, S
,
Spindler, M
Fformat:
Conference item
Cyhoeddwyd:
2002
Daliadau
Disgrifiad
Eitemau Tebyg
Dangos Staff
Disgrifiad
Crynodeb:
Eitemau Tebyg
Creatine kinase knockout mice show left ventricular hypertrophy and dilatation, but unaltered remodeling post-myocardial infarction.
gan: Nahrendorf, M, et al.
Cyhoeddwyd: (2005)
Impaired cardiac contractile function in AGAT knockout mice devoid of creatine is rescued by homoarginine but not creatine
gan: Faller, K, et al.
Cyhoeddwyd: (2017)
Cardiac phenotype of mitochondrial creatine kinase knockout mice is modified on a pure C57BL/6 genetic background.
gan: Lygate, C, et al.
Cyhoeddwyd: (2009)
Dissimilarity in the folding of human cytosolic creatine kinase isoenzymes.
gan: Yin Wang, et al.
Cyhoeddwyd: (2011-01-01)
Mitochondrial creatine kinase is critically necessary for normal myocardial high-energy phosphate metabolism.
gan: Spindler, M, et al.
Cyhoeddwyd: (2002)