Impaired exercise capacity in cytosolic (M-CK) but not in mitochondrial creatine kinase knockout mice

Samankaltaisia teoksia

• Creatine kinase knockout mice show left ventricular hypertrophy and dilatation, but unaltered remodeling post-myocardial infarction.
  Tekijä: Nahrendorf, M, et al.
  Julkaistu: (2005)
• Impaired cardiac contractile function in AGAT knockout mice devoid of creatine is rescued by homoarginine but not creatine
  Tekijä: Faller, K, et al.
  Julkaistu: (2017)
• Cardiac phenotype of mitochondrial creatine kinase knockout mice is modified on a pure C57BL/6 genetic background.
  Tekijä: Lygate, C, et al.
  Julkaistu: (2009)
• Dissimilarity in the folding of human cytosolic creatine kinase isoenzymes.
  Tekijä: Yin Wang, et al.
  Julkaistu: (2011-01-01)
• Mitochondrial creatine kinase is critically necessary for normal myocardial high-energy phosphate metabolism.
  Tekijä: Spindler, M, et al.
  Julkaistu: (2002)