Impaired exercise capacity in cytosolic (M-CK) but not in mitochondrial creatine kinase knockout mice
المؤلفون الرئيسيون: | Maass, A, Bauer, L, Neubauer, S, Spindler, M |
---|---|
التنسيق: | Conference item |
منشور في: |
2002
|
مواد مشابهة
-
Creatine kinase knockout mice show left ventricular hypertrophy and dilatation, but unaltered remodeling post-myocardial infarction.
حسب: Nahrendorf, M, وآخرون
منشور في: (2005) -
Impaired cardiac contractile function in AGAT knockout mice devoid of creatine is rescued by homoarginine but not creatine
حسب: Faller, K, وآخرون
منشور في: (2017) -
Cardiac phenotype of mitochondrial creatine kinase knockout mice is modified on a pure C57BL/6 genetic background.
حسب: Lygate, C, وآخرون
منشور في: (2009) -
Dissimilarity in the folding of human cytosolic creatine kinase isoenzymes.
حسب: Yin Wang, وآخرون
منشور في: (2011-01-01) -
Mitochondrial creatine kinase is critically necessary for normal myocardial high-energy phosphate metabolism.
حسب: Spindler, M, وآخرون
منشور في: (2002)