Impaired exercise capacity in cytosolic (M-CK) but not in mitochondrial creatine kinase knockout mice
Prif Awduron: | Maass, A, Bauer, L, Neubauer, S, Spindler, M |
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Fformat: | Conference item |
Cyhoeddwyd: |
2002
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Eitemau Tebyg
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Creatine kinase knockout mice show left ventricular hypertrophy and dilatation, but unaltered remodeling post-myocardial infarction.
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Impaired cardiac contractile function in AGAT knockout mice devoid of creatine is rescued by homoarginine but not creatine
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Cardiac phenotype of mitochondrial creatine kinase knockout mice is modified on a pure C57BL/6 genetic background.
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Dissimilarity in the folding of human cytosolic creatine kinase isoenzymes.
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Mitochondrial creatine kinase is critically necessary for normal myocardial high-energy phosphate metabolism.
gan: Spindler, M, et al.
Cyhoeddwyd: (2002)