Th17 cells expressing KIR3DL2 and enriched for gut homing markers are increased in ankylosing spondylitis

Th17 cells expressing KIR3DL2 and enriched for gut homing markers are increased in ankylosing spondylitis

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Podrobná bibliografie
Hlavní autoři:	Ridley, A, Kollnberger, S, Wong, I, Shaw, J, Chan, A, Fleming, M, Cummings, F, Bowness, P
Médium:	Conference item
Vydáno:	2011

Podobné jednotky

TH17 CELLS EXPRESSING KIR3DL2 AND ENRICHED FOR GUT HOMING MARKERS ARE INCREASED IN ANKYLOSING SPONDYLITIS
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TH17 CELLS EXPRESSING KIR3DL2 AND ENRICHED FOR GUT HOMING MARKERS ARE INCREASED IN ANKYLOSING SPONDYLITIS
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Th17 cells expressing KIR3DL2+are increased in ankylosing spondylitis
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KIR3DL2 INTERACTION WITH HLA-B27 PROMOTES THE SURVIVAL OF TH17 CELLS IN ANKYLOSING SPONDYLITIS
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Th17 cells expressing KIR3DL2+ and responsive to HLA-B27 homodimers are increased in ankylosing spondylitis.
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Increased IL-23 receptor expression is observed on KIR3DL2+CD4+T cells in Ankylosing Spondylitis and correlates with IL-23R polymorphisms
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KIR3DL2 binds to HLA-B27 dimers and free H chains more strongly than other HLA class I and promotes the expansion of T cells in ankylosing spondylitis.
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The immunology of Ankylosing Spondylitis
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Interaction of HLA-B27 homodimers with KIR3DL1 and KIR3DL2, unlike HLA-B27 heterotrimers, is independent of the sequence of bound peptide.
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Molecular determinants for KIR3DL2 binding to HLA-B27 and other HLA-class I ligands
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Expansion and enhanced survival of natural killer cells expressing the killer immunoglobulin-like receptor KIR3DL2 in spondylarthritis.
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