Beer, N., Tribble, N., Colclough, K., Arundel, P., Grimsby, J., Chik, C., . . . Gloyn, A. (2010). Naturally occurring glucokinase mutations at the same amino acid residue cause opposite clinical phenotypes of hypo- and hyperglycaemia.

Beer, N., N. Tribble, K. Colclough, P. Arundel, J. Grimsby, C. Chik, S. Ellard, and A. Gloyn. Naturally Occurring Glucokinase Mutations at the Same Amino Acid Residue Cause Opposite Clinical Phenotypes of Hypo- and Hyperglycaemia. 2010.

Beer, N., et al. Naturally Occurring Glucokinase Mutations at the Same Amino Acid Residue Cause Opposite Clinical Phenotypes of Hypo- and Hyperglycaemia. 2010.