Impairment of cholinergic signaling reduces atrial beta-adrenergic responsiveness by enhancing eNOS expression

Impairment of cholinergic signaling reduces atrial beta-adrenergic responsiveness by enhancing eNOS expression

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Zhang, Y, Danson, E, Sears, C, Edwards, A, Casadei, B, Paterson, D
التنسيق:	Conference item
منشور في:	2004

مواد مشابهة

Disruption of inhibitory G-proteins mediates a reduction in atrial beta-adrenergic signaling by enhancing eNOS expression.
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nNOS V eNOS mediated regulation of beta adrenergic inotropic and lusitropic responses in left ventricular myocytes
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Are myocardial eNOS and nNOS involved in the beta-adrenergic and muscarinic regulation of inotropy? A systematic investigation.
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Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left-ventricular contraction and relaxation in vivo
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Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left ventricular contraction and relaxation in vivo
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Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes
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Reduced expression of eNOS in the fractured femoral neck
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Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1
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Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
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The negative inotropic effect of beta(3)-adrenergic receptor stimulation in nNOS(-/-) mice is restored by oxypurinol
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The Cholinergic and Adrenergic Autocrine Signaling Pathway Mediates Immunomodulation in Oyster Crassostrea gigas
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Sympathetic activation increases NO release from eNOS but neither eNOS nor nNOS play an essential role in exercise hyperemia in the human forearm.
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Cholinergic control of heart rate by nitric oxide is site specific.
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Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
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Beta adrenergic blockade reduces utilitarian judgement.
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Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
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mTOR contributes to endothelium-dependent vasorelaxation by promoting eNOS expression and preventing eNOS uncoupling
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Participation of cholinergic and adrenergic mechanisms in gastric reactions to exogenous serotonin infusion
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Pupil fluctuations track rapid changes in adrenergic and cholinergic activity in cortex
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Expression of eNOS in kidneys from hypertensive patients
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Cardiac cholinergic NO-cGMP signaling following acute myocardial infarction and nNOS gene transfer.
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Orientin Reduces Myocardial Infarction Size via eNOS/NO Signaling and Thus Mitigates Adverse Cardiac Remodeling
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High-fat stimulation induces atrial neural remodeling by reducing NO production via the CRIF1/eNOS/P21 axi
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The roles of beta-adrenergic receptors in tumorigenesis and the possible use of beta-adrenergic blockers for cancer treatment: possible genetic and cell-signaling mechanisms
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Myricetin ameliorated ischemia/reperfusion-induced brain endothelial permeability by improvement of eNOS uncoupling and activation eNOS/NO
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Beta adrenergic blockers poisoning
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eNOS in congenital to adult cardiovascular disease
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Vascular nitric oxide: Beyond eNOS
حسب: Yingzi Zhao, وآخرون
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PKC-dependent phosphorylation of eNOS at T495 regulates eNOS coupling and endothelial barrier function in response to G+ -toxins.
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Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
حسب: Bendall, J, وآخرون
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Effects of inhibition of eNOS and nNOS on forearm blood flow during reflex sympathetic activation at rest and during exercise
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Loss of cholinergic innervation differentially affects eNOS-mediated blood flow, drainage of Aβ and cerebral amyloid angiopathy in the cortex and hippocampus of adult mice
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Neural control of coronary artery blood flow by non‐adrenergic and non‐cholinergic mechanisms
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Tetrahydrobiopterin- and glutathionylation-dependent regulation of eNOS coupling: Relationships revealed by eNOS glutathionylation mutants in cells with tet-regulated GTP cyclohydrolase I expression
حسب: Crabtree, M, وآخرون
منشور في: (2012)