Bone marrow-derived haematopoietic and mesenchymal stem cells colonise kidney but fail to produce collagen I in renal fibrosis

Bone marrow-derived haematopoietic and mesenchymal stem cells colonise kidney but fail to produce collagen I in renal fibrosis

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Roufosse, C, Alexakis, C, Bou-Gharios, G, Otto, W, Prodromidi, E, Alter, J, Jeffery, R, Cook, H, Poulsom, R
Μορφή:	Conference item
Έκδοση:	2006

Παρόμοια τεκμήρια

Bone marrow-derived cells do not contribute significantly to collagen I synthesis in a murine model of renal fibrosis.
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Bone marrow-derived cells participate in renal regeneration but make a trivial contribution to collagen I synthesis in a murine model of kidney fibrosis
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Bone marrow stem cells contribution to tissue fibrosis
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The bone marrow functionally contributes to liver fibrosis.
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Col1a2 enhancer regulates collagen activity during development and in adult tissue repair.
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Negligible contribution of bone marrow-derived cells to collagen production during hepatic fibrogenesis in mice.
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Failure of bone marrow stem cell therapy to improve liver fibrosis in the CCl4 murine model
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Haematopoietic stem/progenitor cell interactions with the bone marrow vascular niche
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Differential contribution of dermal resident and bone marrow-derived cells to collagen production during wound healing and fibrogenesis in mice.
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Distant mesenchymal progenitors contribute to skin wound healing and produce collagen: evidence from a murine fetal microchimerism model.
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The Coloniser and the Colonised: Reflections on Translation as Contested Space
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Identification of a dual-function peptide for the detection and haematopoietic potency assessment of type I collagen hydrolysates
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Effects of T-lymphocyte depletion on muscle fibrosis in the mdx mouse.
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Cell type-specific intervention of transforming growth factor beta/Smad signaling suppresses collagen gene expression and hepatic fibrosis in mice.
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Regulation of collagen type I in vascular smooth muscle cells by competition between Nkx2.5 and deltaEF1/ZEB1.
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High doses of TGF-β potently suppress type I collagen via the transcription factor CUX1.
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Wound-associated macrophages control collagen 1α2 transcription during the early stages of skin wound healing.
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