Aldehyde dehydrogenase-2 inhibition blocks remote preconditioning in experimental and human models.
Mitochondrial aldehyde dehydrogenase-2 (ALDH-2) is involved in preconditioning pathways, but its role in remote ischaemic preconditioning (rIPC) is unknown. We investigated its role in animal and human models of rIPC. (i) In a rabbit model of myocardial infarction, rIPC alone reduced infarct size [6...
المؤلفون الرئيسيون: | Contractor, H, Støttrup, N, Cunnington, C, Manlhiot, C, Diesch, J, Ormerod, J, Jensen, R, Bøtker, H, Redington, A, Schmidt, MR, Ashrafian, H, Kharbanda, R |
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التنسيق: | Journal article |
اللغة: | English |
منشور في: |
2013
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مواد مشابهة
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Aldehyde dehydrogenase-2 inhibition blocks remote preconditioning in experimental and human models
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Aldehyde dehydrogenase-2 is necessary for remote preconditioning
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Remote ischemic preconditioning impairs ventricular function and increases infarct size after prolonged ischemia in the isolated neonatal rabbit heart
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Remote ischemic preconditioning impairs ventricular function and increases infarct size after prolonged ischemia in the isolated neonatal rabbit heart.
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Remote ischemic preconditioning with - but not without - metabolic support protects the neonatal porcine heart against ischemia-reperfusion injury
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