Calcium-activated potassium channel SK1- and IK1-like immunoreactivity in injured human sensory neurones and its regulation by neurotrophic factors.
Calcium-activated potassium ion channels SK and IK (small and intermediate conductance, respectively) may be important in the pathophysiology of pain following nerve injury, as SK channels are known to impose a period of reduced excitability after each action potential by afterhyperpolarization. We...
Main Authors: | Boettger, M, Till, S, Chen, M, Anand, U, Otto, W, Plumpton, C, Trezise, D, Tate, SN, Bountra, C, Coward, K, Birch, R, Anand, P |
---|---|
格式: | Journal article |
语言: | English |
出版: |
2002
|
相似书籍
-
Decreased potassium channel IK1 and its regulator neurotrophin-3 (NT-3) in inflamed human bowel.
由: Arnold, S, et al.
出版: (2003) -
Plasticity of TTX-sensitive sodium channels PN1 and brain III in injured human nerves.
由: Coward, K, et al.
出版: (2001) -
Sodium channel Nav1.8 immunoreactivity in painful human dental pulp.
由: Renton, T, et al.
出版: (2005) -
Sodium channel beta1 and beta2 subunits parallel SNS/PN3 alpha-subunit changes in injured human sensory neurons.
由: Coward, K, et al.
出版: (2001) -
Increased sodium channel Na(v)1.8 immunoreactivity in painful human dental pulp
由: Renton, T, et al.
出版: (2004)