Interstitial fibrosis is associated with increased COL1A2 transcription in AA-injured renal tubular epithelial cells in vivo.
Accumulation of type I collagen is a key event in renal interstitial fibrosis. As there is no effective treatment, understanding the site where collagen is transcribed and the factors driving it in response to disease in vivo is critical for designing future therapies. The present research investiga...
Κύριοι συγγραφείς: | Fragiadaki, M, Witherden, A, Kaneko, T, Sonnylal, S, Pusey, C, Bou-Gharios, G, Mason, R |
---|---|
Μορφή: | Journal article |
Γλώσσα: | English |
Έκδοση: |
2011
|
Παρόμοια τεκμήρια
Παρόμοια τεκμήρια
-
High doses of TGF-β potently suppress type I collagen via the transcription factor CUX1.
ανά: Fragiadaki, M, κ.ά.
Έκδοση: (2011) -
Fibroblast-directed CTGF over-expression in vivo promotes systemic connective tissue fibrosis reminiscent of fibroproliferative diseases
ανά: Sonnylal, S, κ.ά.
Έκδοση: (2009) -
Norcantharidin inhibits renal interstitial fibrosis by blocking the tubular epithelial-mesenchymal transition.
ανά: Ying Li, κ.ά.
Έκδοση: (2013-01-01) -
Correction: Norcantharidin Inhibits Renal Interstitial Fibrosis by Blocking the Tubular Epithelial-Mesenchymal Transition
Έκδοση: (2013-01-01) -
Fluorofenidone inhibits apoptosis of renal tubular epithelial cells in rats with renal interstitial fibrosis
ανά: Hui Yang, κ.ά.
Έκδοση: (2019-10-01)