Monocyte-derived tumour necrosis factor stimulated gene-6 (TSG-6) alters bone mass via effects on osteoblasts and osteoclasts

Monocyte-derived tumour necrosis factor stimulated gene-6 (TSG-6) alters bone mass via effects on osteoblasts and osteoclasts

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Afrough, S, Mahoney, D, Sabokbar, A, Horwood, N
Μορφή:	Conference item
Έκδοση:	2012

Παρόμοια τεκμήρια

TSG-6 regulates bone remodeling through inhibition of osteoblastogenesis and osteoclast activation.
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TSG-6 regulates bone remodelling through inhibition of osteoblastogenesis and osteoclast activation
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TSG-6 inhibits osteoclast activity via an autocrine mechanism and is functionally synergistic with osteoprotegerin
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The role of tumour necrosis factor alpha stimulated gene-6 on bone
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TSG-6 Acts Synergisticly with OPG to Inhibit Bone Resorption
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TSG-6, new regulator of bone remodelling
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TSG-6: a new regulator of bone remodelling
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TSG-6 inhibits osteoclast activity via an autocrine mechanism and is functionally synergistic with osteoprotegerin.
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TSG-6 inhibits osteoclast activity via an autocrine mechanism and is functionally synergistic with osteoprotegerin.
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Human osteoblasts support human monocyte-osteoclast differentiation: Evidence of prostaglandin stimulation
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Human osteoblasts support human monocyte-osteoclast differentiation
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The effect of interleukin-1β, tumour necrosis factor α and particle phagocytosis on monocyte-osteoclast differentiation
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TSG-6: AN AUTOCRINE REGULATOR OF INFLAMMATORY JOINT DISEASE?
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Plasticity of the TSG-6 HA-binding loop and mobility in the TSG-6-HA complex revealed by NMR and X-ray crystallography.
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TSG-6 modulates the interaction between hyaluronan and cell surface CD44.
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Brain Ischemic Tolerance Triggered by Preconditioning Involves Modulation of Tumor Necrosis Factor-α-Stimulated Gene 6 (TSG-6) in Mice Subjected to Transient Middle Cerebral Artery Occlusion
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