Dysregulated hematopoietic stem and progenitor cell activity promotes interleukin-23-driven chronic intestinal inflammation.
In interleukin-23 (IL-23)-dependent colitis, there is excessive accumulation of short-lived neutrophils and inflammatory monocytes in the intestine. It is unknown whether this reflects changes in mature cell populations or whether the IL-23-driven colitogenic T cell program regulates upstream hemato...
Prif Awduron: | Griseri, T, McKenzie, B, Schiering, C, Powrie, F |
---|---|
Fformat: | Conference item |
Cyhoeddwyd: |
2012
|
Eitemau Tebyg
-
Dysregulated Hematopoietic Stem and Progenitor Cell Activity Promotes Interleukin-23-Driven Chronic Intestinal Inflammation
gan: Griseri, T, et al.
Cyhoeddwyd: (2012) -
Exacerbated hematopoietic stem and progenitor cell activity during intestinal inflammation
gan: Griseri, T, et al.
Cyhoeddwyd: (2011) -
Interleukin-23 drives intestinal inflammation through direct activity on T cells
gan: Schiering, C, et al.
Cyhoeddwyd: (2010) -
Interleukin-23 drives intestinal inflammation through direct activity on T cells
gan: Ahern, P, et al.
Cyhoeddwyd: (2011) -
Interleukin-23 drives intestinal inflammation through direct activity on T cells.
gan: Ahern, P, et al.
Cyhoeddwyd: (2010)